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Inge Grundke-Iqbal, PhD

Affiliations: 
Neurochemistry New York State Institute Basic Research in Developmental Disabilities, NY, United States 
Area:
Alzheimer disease
Google:
"Inge Grundke-Iqbal"
Bio:

http://www.researchprofiles.collexis.com/jad/expert.asp?u_id=90

http://www.biomedexperts.com/Profile.bme/500032/Inge_Grundke-Iqbal

Cross-listing: Neurotree - Neuropathology Tree

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Khalid Iqbal collaborator New York State Institute Basic Research in Developmental Disabilities
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Publications

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Wang X, Blanchard J, Grundke-Iqbal I, et al. (2015) Memantine Attenuates Alzheimer's Disease-Like Pathology and Cognitive Impairment. Plos One. 10: e0145441
Kazim SF, Cardenas-Aguayo Mdel C, Arif M, et al. (2015) Sera from children with autism induce autistic features which can be rescued with a CNTF small peptide mimetic in rats. Plos One. 10: e0118627
Wang X, Blanchard J, Tung YC, et al. (2015) Inhibition of Protein Phosphatase-2A (PP2A) by I1PP2A Leads to Hyperphosphorylation of Tau, Neurodegeneration, and Cognitive Impairment in Rats. Journal of Alzheimer's Disease : Jad. 45: 423-35
Dai CL, Chen X, Kazim SF, et al. (2015) Passive immunization targeting the N-terminal projection domain of tau decreases tau pathology and improves cognition in a transgenic mouse model of Alzheimer disease and tauopathies. Journal of Neural Transmission (Vienna, Austria : 1996). 122: 607-17
Arif M, Wei J, Zhang Q, et al. (2014) Cytoplasmic retention of protein phosphatase 2A inhibitor 2 (I2PP2A) induces Alzheimer-like abnormal hyperphosphorylation of Tau. The Journal of Biological Chemistry. 289: 27677-91
Arif M, Kazim SF, Grundke-Iqbal I, et al. (2014) Tau pathology involves protein phosphatase 2A in parkinsonism-dementia of Guam. Proceedings of the National Academy of Sciences of the United States of America. 111: 1144-9
Wang X, Blanchard J, Grundke-Iqbal I, et al. (2014) Alzheimer disease and amyotrophic lateral sclerosis: an etiopathogenic connection. Acta Neuropathologica. 127: 243-56
Basurto-Islas G, Grundke-Iqbal I, Tung YC, et al. (2013) Activation of asparaginyl endopeptidase leads to Tau hyperphosphorylation in Alzheimer disease. The Journal of Biological Chemistry. 288: 17495-507
Jin N, Qian W, Yin X, et al. (2013) CREB regulates the expression of neuronal glucose transporter 3: a possible mechanism related to impaired brain glucose uptake in Alzheimer's disease. Nucleic Acids Research. 41: 3240-56
Cardenas-Aguayo Mdel C, Kazim SF, Grundke-Iqbal I, et al. (2013) Neurogenic and neurotrophic effects of BDNF peptides in mouse hippocampal primary neuronal cell cultures. Plos One. 8: e53596
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