Maya K. Desai, Ph.D.
Affiliations: | Stanford University, Palo Alto, CA |
Area:
Alzheimer, myelin, oligodendrocytesGoogle:
"Maya Desai"Cross-listing: Neurotree
Parents
Sign in to add mentorWilliam J. Bowers | grad student | 2005-2011 | Rochester | |
(The Genesis of Region-specific White Matter Deficits During Early Alzheimer's Disease Pathogenesis.) |
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Publications
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Desai MK, Guercio BJ, Narrow WC, et al. (2011) An Alzheimer's disease-relevant presenilin-1 mutation augments amyloid-beta-induced oligodendrocyte dysfunction. Glia. 59: 627-40 |
Desai MK, Mastrangelo MA, Ryan DA, et al. (2010) Early oligodendrocyte/myelin pathology in Alzheimer's disease mice constitutes a novel therapeutic target. The American Journal of Pathology. 177: 1422-35 |
Bowers WJ, Desai MK, Mastrangelo MA, et al. (2010) P2-242: Oligodendrocyte cell lineage represents an amyloid beta 1-42 sensitive cell population leading to white matter alterations during early Alzheimer's disease Alzheimer's & Dementia. 6: S385-S385 |
Desai MK, Sudol KL, Janelsins MC, et al. (2009) Triple-transgenic Alzheimer's disease mice exhibit region-specific abnormalities in brain myelination patterns prior to appearance of amyloid and tau pathology. Glia. 57: 54-65 |
Desai MK, Sudol KL, Janelsins MC, et al. (2008) P3-404: Triple-transgenic Alzheimer's disease mice exhibit marked abnormalities in brain myelination patterns prior to appearance of amyloid and tau pathology Alzheimer's & Dementia. 4: T640-T640 |