Leonard Petrucelli, PhD
Affiliations: | Neuroscience Research | Mayo Clinic Jacksonville, Jacksonville, FL, United States |
Area:
http://www.biomedexperts.com/Profile.bme/1110907/Leonard_PetrucelliWebsite:
http://mayoresearch.mayo.edu/mayo/research/staff/petrucelli_l.cfmGoogle:
"Leonard Petrucelli"Bio:
http://mayoresearch.mayo.edu/petrucelli_lab/
http://www.alzforum.org/com/res/detail.asp?id={6FC44417-BF30-4883-A2A1-95E498E8F852}
http://www.molecularneurodegeneration.com/edboard/start.asp?id=730361
Cross-listing: Neurotree
Children
Sign in to add trainee| Joseph W Paul | research assistant | 2011-2013 | Mayo Clinc (Cell Biology Tree) |
| Mercedes Prudencio | post-doc | Mayo Clinic Jacksonville (Neurotree) |
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Publications
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| Li Z, Weller CA, Shah S, et al. (2024) ProtPipe: A Multifunctional Data Analysis Pipeline for Proteomics and Peptidomics. Genomics, Proteomics & Bioinformatics |
| Cruz-González G, Meschia JF, Madden BJ, et al. (2024) Recent advances in proteomic analysis to study carotid artery plaques. Jvs-Vascular Science. 5: 100215 |
| Calliari A, Daughrity LM, Albagli EA, et al. (2024) Correction: HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases. Molecular Neurodegeneration. 19: 56 |
| Castellanos Otero P, Todd TW, Shao W, et al. (2024) Generation and characterization of monoclonal antibodies against pathologically phosphorylated TDP-43. Plos One. 19: e0298080 |
| Babu S, Nicholson KA, Rothstein JD, et al. (2024) Apilimod dimesylate in C9orf72 amyotrophic lateral sclerosis: a randomized phase 2a clinical trial. Brain : a Journal of Neurology |
| Calliari A, Daughrity LM, Albagli EA, et al. (2024) HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases. Molecular Neurodegeneration. 19: 29 |
| Erben Y, Prudencio M, Marquez CP, et al. (2024) Erratum: Neurofilament light chain and vaccination status associate with clinical outcomes in severe COVID-19. Iscience. 27: 109501 |
| Zeng Y, Lovchykova A, Akiyama T, et al. (2024) TDP-43 nuclear loss in FTD/ALS causes widespread alternative polyadenylation changes. Biorxiv : the Preprint Server For Biology |
| Seddighi S, Qi YA, Brown AL, et al. (2024) Mis-spliced transcripts generate de novo proteins in TDP-43-related ALS/FTD. Science Translational Medicine. eadg7162 |
| Marks JD, Ayuso VE, Carlomagno Y, et al. (2024) TMEM106B core deposition associates with TDP-43 pathology and is increased in risk SNP carriers for frontotemporal dementia. Science Translational Medicine. 16: eadf9735 |