Year |
Citation |
Score |
2017 |
Chang NH, Manion KP, Loh C, Pau E, Baglaenko Y, Wither JE. Multiple tolerance defects contribute to the breach of B cell tolerance in New Zealand Black chromosome 1 congenic mice. Plos One. 12: e0179506. PMID 28628673 DOI: 10.1371/Journal.Pone.0179506 |
0.813 |
|
2016 |
Baglaenko Y, Manion KP, Chang NH, Gracey E, Loh C, Wither JE. IL-10 Production Is Critical for Sustaining the Expansion of CD5+ B and NKT Cells and Restraining Autoantibody Production in Congenic Lupus-Prone Mice. Plos One. 11: e0150515. PMID 26964093 DOI: 10.1371/Journal.Pone.0150515 |
0.787 |
|
2015 |
Schaffert SA, Loh C, Wang S, Arnold CP, Axtell RC, Newell EW, Nolan G, Ansel KM, Davis MM, Steinman L, Chen CZ. mir-181a-1/b-1 Modulates Tolerance through Opposing Activities in Selection and Peripheral T Cell Function. Journal of Immunology (Baltimore, Md. : 1950). PMID 26163591 DOI: 10.4049/Jimmunol.1401587 |
0.301 |
|
2015 |
Baglaenko Y, Manion KP, Chang NH, Loh C, Lajoie G, Wither JE. Suppression of autoimmunity by CD5(+) IL-10-producing B cells in lupus-prone mice. Genes and Immunity. 16: 311-20. PMID 25973757 DOI: 10.1038/gene.2015.17 |
0.721 |
|
2013 |
Pau E, Loh C, Minty GE, Chang NH, Wither JE. Identification of a lupus-susceptibility locus leading to impaired clearance of apoptotic debris on New Zealand Black chromosome 13. Genes and Immunity. 14: 154-61. PMID 23328841 DOI: 10.1038/Gene.2012.64 |
0.809 |
|
2012 |
Pau E, Cheung YH, Loh C, Lajoie G, Wither JE. TLR tolerance reduces IFN-alpha production despite plasmacytoid dendritic cell expansion and anti-nuclear antibodies in NZB bicongenic mice. Plos One. 7: e36761. PMID 22574220 DOI: 10.1371/Journal.Pone.0036761 |
0.799 |
|
2011 |
Loh C, Pau E, Lajoie G, Li TT, Baglaenko Y, Cheung YH, Chang NH, Wither JE. Epistatic suppression of fatal autoimmunity in New Zealand black bicongenic mice. Journal of Immunology (Baltimore, Md. : 1950). 186: 5845-53. PMID 21464090 DOI: 10.4049/Jimmunol.1003426 |
0.819 |
|
2011 |
Pau E, Chang NH, Loh C, Lajoie G, Wither JE. Abrogation of pathogenic IgG autoantibody production in CD40L gene-deleted lupus-prone New Zealand Black mice. Clinical Immunology (Orlando, Fla.). 139: 215-27. PMID 21414847 DOI: 10.1016/J.Clim.2011.02.005 |
0.803 |
|
2011 |
Loh C, Pau E, Chang NH, Wither JE. An intrinsic B-cell defect supports autoimmunity in New Zealand black chromosome 13 congenic mice. European Journal of Immunology. 41: 527-36. PMID 21268021 DOI: 10.1002/Eji.201040983 |
0.809 |
|
2010 |
Chang NH, Cheung YH, Loh C, Pau E, Roy V, Cai YC, Wither J. B Cell activating factor (BAFF) and T cells cooperate to breach B cell tolerance in lupus-prone New Zealand Black (NZB) mice Plos One. 5. PMID 20661465 DOI: 10.1371/Journal.Pone.0011691 |
0.811 |
|
2009 |
Cheung YH, Loh C, Pau E, Kim J, Wither J. Insights into the genetic basis and immunopathogenesis of systemic lupus erythematosus from the study of mouse models Seminars in Immunology. 21: 372-382. PMID 19926489 DOI: 10.1016/J.Smim.2009.10.005 |
0.728 |
|
2007 |
Loh C, Cai YC, Bonventi G, Lajoie G, Macleod R, Wither JE. Dissociation of the genetic loci leading to b1a and NKT cell expansions from autoantibody production and renal disease in B6 mice with an introgressed New Zealand Black chromosome 4 interval. Journal of Immunology (Baltimore, Md. : 1950). 178: 1608-17. PMID 17237410 DOI: 10.4049/Jimmunol.178.3.1608 |
0.799 |
|
2005 |
Wither JE, Loh C, Lajoie G, Heinrichs S, Cai YC, Bonventi G, MacLeod R. Colocalization of expansion of the splenic marginal zone population with abnormal B cell activation and autoantibody production in B6 mice with an introgressed New Zealand Black chromosome 13 interval. Journal of Immunology (Baltimore, Md. : 1950). 175: 4309-19. PMID 16177071 DOI: 10.4049/Jimmunol.175.7.4309 |
0.842 |
|
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