Edward M. Campbell
Affiliations: | Cell Biology, Neurobiology and Anatomy | Loyola University Chicago, Chicago, IL, United States |
Area:
Cell Biology, Virology BiologyGoogle:
"Edward Campbell"Children
Sign in to add traineeJayalaxmi Sastri | grad student | 2013 | Loyola University Chicago |
Zana Lukic | grad student | 2014 | Loyola University Chicago |
Collaborators
Sign in to add collaboratorFletcher A. White | collaborator | 2013- | Loyola Univ Chicago, Stritch Sch Medicine (Neurotree) |
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Publications
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Badieyan S, Lichon D, Andreas MP, et al. (2023) HIV-1 binds dynein directly to hijack microtubule transport machinery. Biorxiv : the Preprint Server For Biology |
Nguyen T, Nguyen N, Cochran AG, et al. (2023) Repeated closed-head mild traumatic brain injury-induced inflammation is associated with nociceptive sensitization. Journal of Neuroinflammation. 20: 196 |
Dharan A, Campbell EM. (2022) Teaching old dogmas new tricks: recent insights into the nuclear import of HIV-1. Current Opinion in Virology. 53: 101203 |
Al-Harthi L, Campbell EM, Schneider JA, et al. (2020) What HIV in the brain can teach us about SARS-CoV-2 neurologic complications? Aids Research and Human Retroviruses |
Taylor HE, Calantone N, Lichon D, et al. (2020) mTOR Overcomes Multiple Metabolic Restrictions to Enable HIV-1 Reverse Transcription and Intracellular Transport. Cell Reports. 31: 107810 |
Dharan A, Bachmann N, Talley S, et al. (2020) Nuclear pore blockade reveals that HIV-1 completes reverse transcription and uncoating in the nucleus. Nature Microbiology |
Kim K, Dauphin A, Komurlu S, et al. (2019) Cyclophilin A protects HIV-1 from restriction by human TRIM5α. Nature Microbiology |
Talley S, Kalinina O, Winek M, et al. (2019) A Novel Caspase-1 Biosensor to Monitor the Progression of Inflammation In Vivo. Journal of Immunology (Baltimore, Md. : 1950) |
Imam S, Kömürlü S, Mattick J, et al. (2019) K63-linked Ubiquitin is Required for Restriction of HIV-1 Reverse Transcription and Capsid Destabilization by Rhesus Trim5alpha. Journal of Virology |
Kömürlü S, Bradley M, Smolin N, et al. (2019) Defects in assembly explain reduced antiviral activity of the G249D polymorphism in human TRIM5α. Plos One. 14: e0212888 |