1989-1994 CELLULAR BIOLOGY AND ANATOMY University of Kansas School of Medicine, Kansas City, KS, United States 
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Muresan V, Ladescu Muresan Z. (2016) Shared Molecular Mechanisms in Alzheimer's Disease and Amyotrophic Lateral Sclerosis: Neurofilament-Dependent Transport of sAPP, FUS, TDP-43 and SOD1, with Endoplasmic Reticulum-Like Tubules. Neuro-Degenerative Diseases. 16: 55-61
Muresan V, Ladescu Muresan Z. (2015) Amyloid-β precursor protein: Multiple fragments, numerous transport routes and mechanisms. Experimental Cell Research. 334: 45-53
Villegas C, Muresan V, Ladescu Muresan Z. (2014) Dual-tagged amyloid-β precursor protein reveals distinct transport pathways of its N- and C-terminal fragments. Human Molecular Genetics. 23: 1631-43
Muresan V, Villegas C, Ladescu Muresan Z. (2014) Functional interaction between amyloid-β precursor protein and peripherin neurofilaments: a shared pathway leading to Alzheimer's disease and amyotrophic lateral sclerosis? Neuro-Degenerative Diseases. 13: 122-5
Muresan V, Muresan Z. (2012) Unconventional functions of microtubule motors. Archives of Biochemistry and Biophysics. 520: 17-29
Muresan V, Muresan Z. (2012) A persistent stress response to impeded axonal transport leads to accumulation of amyloid-β in the endoplasmic reticulum, and is a probable cause of sporadic Alzheimer's disease. Neuro-Degenerative Diseases. 10: 60-3
Muresan V, Muresan Z. (2009) Is abnormal axonal transport a cause, a contributing factor or a consequence of the neuronal pathology in Alzheimer's disease? Future Neurology. 4: 761-773
Muresan V, Varvel NH, Lamb BT, et al. (2009) The cleavage products of amyloid-beta precursor protein are sorted to distinct carrier vesicles that are independently transported within neurites. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 29: 3565-78
Muresan V, Muresan Z. (2008) No conventional function for the conventional kinesin? Traffic (Copenhagen, Denmark). 9: 1823-7
Muresan Z, Muresan V. (2008) Seeding neuritic plaques from the distance: a possible role for brainstem neurons in the development of Alzheimer's disease pathology. Neuro-Degenerative Diseases. 5: 250-3
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