Stephen C. Cannon - Publications

Affiliations: 
2015- Physiology David Geffen School of Medicine at UCLA, Los Angeles, CA, United States 
Area:
channelopathies, skeletal muscle, sodium channels
Website:
https://cannonlab.dgsom.ucla.edu/pages/

112 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2023 Quiñonez M, DiFranco M, Wu F, Cannon SC. Retigabine suppresses loss of force in mouse models of hypokalaemic periodic paralysis. Brain : a Journal of Neurology. PMID 36718088 DOI: 10.1093/brain/awac441  0.714
2022 DiFranco M, Cannon S. Voltage-Dependent Ca Release Is Impaired in Hypokalemic Periodic Paralysis Caused by Ca1.1-R528H but not by Na1.4-R669H. American Journal of Physiology. Cell Physiology. PMID 35759432 DOI: 10.1152/ajpcell.00209.2022  0.36
2021 Savalli N, Angelini M, Steccanella F, Wier J, Wu F, Quinonez M, DiFranco M, Neely A, Cannon SC, Olcese R. The distinct role of the four voltage sensors of the skeletal CaV1.1 channel in voltage-dependent activation. The Journal of General Physiology. 153. PMID 34546289 DOI: 10.1085/jgp.202112915  0.763
2021 Wu F, Quinonez M, Cannon SC. Gating pore currents occur in CaV1.1 domain III mutants associated with HypoPP. The Journal of General Physiology. 153. PMID 34463712 DOI: 10.1085/jgp.202112946  0.748
2021 Cannon SC. Epilepsy channelopathies go neddy: stabilizing NaV1.1 channels by neddylation. The Journal of Clinical Investigation. 131. PMID 33855971 DOI: 10.1172/JCI148370  0.354
2020 Kubota T, Wu F, Vicart S, Nakaza M, Sternberg D, Watanabe D, Furuta M, Kokunai Y, Abe T, Kokubun N, Fontaine B, Cannon SC, Takahashi MP. Hypokalaemic periodic paralysis with a charge-retaining substitution in the voltage sensor. Brain Communications. 2: fcaa103. PMID 33005891 DOI: 10.1093/braincomms/fcaa103  0.81
2020 Cannon SC. A role for external Ca2+ in maintaining muscle contractility in periodic paralysis. The Journal of General Physiology. 152. PMID 32463879 DOI: 10.1085/Jgp.202012615  0.373
2020 Stunnenberg B, LoRusso S, Arnold WD, Barohn RJ, Cannon SC, Fontaine B, Griggs RC, Hanna MG, Matthews E, Meola G, Sansone VA, Trivedi JR, van Engelen B, Vicart S, Statland JM. Guidelines on clinical presentation and management of non-dystrophic myotonias. Muscle & Nerve. PMID 32270509 DOI: 10.1002/Mus.26887  0.481
2020 Elia N, Nault T, McMillan HJ, Graham GE, Huang L, Cannon SC. Corrigendum: Myotonic Myopathy With Secondary Joint and Skeletal Anomalies From the c.2386C>G, p.L796V Mutation in . Frontiers in Neurology. 11: 181. PMID 32265824 DOI: 10.3389/Fneur.2020.00181  0.76
2020 Elia N, Nault T, McMillan HJ, Graham GE, Huang L, Cannon SC. Myotonic Myopathy With Secondary Joint and Skeletal Anomalies From the c.2386C>G, p.L769V Mutation in . Frontiers in Neurology. 11: 77. PMID 32117035 DOI: 10.3389/Fneur.2020.00077  0.814
2019 DiFranco M, Quinonez M, Dziedzic RM, Spokoyny AM, Cannon SC. A highly-selective chloride microelectrode based on a mercuracarborand anion carrier. Scientific Reports. 9: 18860. PMID 31827130 DOI: 10.1038/S41598-019-54885-6  0.325
2019 Mason ER, Wu F, Patel RR, Xiao Y, Cannon SC, Cummins TR. Resurgent and Gating Pore Currents Induced by de novo Epilepsy Mutations. Eneuro. PMID 31558572 DOI: 10.1523/ENEURO.0141-19.2019  0.782
2019 Heiny JA, Cannon SC, DiFranco M. A four-electrode method to study dynamics of ion activity and transport in skeletal muscle fibers. The Journal of General Physiology. PMID 31320390 DOI: 10.1085/Jgp.201912398  0.322
2019 Elia N, Palmio J, Castañeda MS, Shieh PB, Quinonez M, Suominen T, Hanna MG, Männikkö R, Udd B, Cannon SC. Myasthenic congenital myopathy from recessive mutations at a single residue in Na1.4. Neurology. PMID 30824560 DOI: 10.1212/Wnl.0000000000007185  0.806
2019 Mi W, Wu F, Quinonez M, DiFranco M, Cannon SC. Recovery from acidosis is a robust trigger for loss of force in murine hypokalemic periodic paralysis. The Journal of General Physiology. PMID 30733232 DOI: 10.1085/Jgp.201812231  0.72
2019 Savalli N, Wu F, Quinonez M, Cannon SC, Olcese R. A Mutation Linked to Malignant Hyperthermia in the Skeletal CaV1.1 Channel Stabilizes the Resting State of Voltage Sensor I and Impairs Channel Activation Biophysical Journal. 116: 111a. DOI: 10.1016/J.Bpj.2018.11.629  0.78
2019 Elia N, Mokhonova E, Quinonez M, Cannon S. A Skeletal Muscle Conditional KCNJ2 Knock-Out Mouse Model for Periodic Paralysis in Andersen-Tawil Syndrome Biophysical Journal. 116: 242a. DOI: 10.1016/J.Bpj.2018.11.1325  0.384
2018 Cannon SC. Skeletal muscle channelopathy: a new risk for sudden infant death syndrome. Lancet (London, England). PMID 29605428 DOI: 10.1016/S0140-6736(18)30477-X  0.325
2018 Lin MA, Cannon SC, Papazian DM. Kv4.2 autism and epilepsy mutation enhances inactivation of closed channels but impairs access to inactivated state after opening. Proceedings of the National Academy of Sciences of the United States of America. PMID 29581270 DOI: 10.1073/Pnas.1717082115  0.531
2018 Wu F, Quinonez M, DiFranco M, Cannon SC. Stac3 enhances expression of human CaV1.1 in Xenopus oocytes and reveals gating pore currents in HypoPP mutant channels. The Journal of General Physiology. PMID 29386226 DOI: 10.1085/Jgp.201711962  0.808
2018 Elia N, Shieh P, Quinonez M, Cannon S. NaV1.4 Loss of Function Changes for Recessively Inherited Myopathy with Fluctuating Weakness Biophysical Journal. 114: 632a. DOI: 10.1016/J.Bpj.2017.11.3417  0.315
2018 Heiny JA, Cannon SC, Di Franco M. A Four Microelectrode Method to Study Intracellular Ion Concentration and Transport in Skeletal Muscle Fibers Biophysical Journal. 114: 627a. DOI: 10.1016/J.Bpj.2017.11.3386  0.332
2018 Savalli N, Wu F, Quinonez M, Cannon SC, Olcese R. Optically-Tracked Structural Rearrangements of the Voltage Sensing Domains in the Human CaV1.1 Channel Biophysical Journal. 114: 39a. DOI: 10.1016/J.Bpj.2017.11.265  0.724
2017 Cannon SC. An atypical CaV1.1 mutation reveals a common mechanism for hypokalemic periodic paralysis. The Journal of General Physiology. PMID 29138267 DOI: 10.1085/Jgp.201711923  0.45
2017 Statland JM, Fontaine B, Hanna MG, Johnson N, Kissel JT, Sansone VA, Shieh PB, Tawil RN, Trivedi J, Cannon SC, Griggs RC. A Review of the Diagnosis and Treatment of Periodic Paralysis. Muscle & Nerve. PMID 29125635 DOI: 10.1002/Mus.26009  0.43
2017 Cannon SC. Sodium Channelopathies of Skeletal Muscle. Handbook of Experimental Pharmacology. PMID 28939973 DOI: 10.1007/164_2017_52  0.607
2017 Cannon SC. Mind the magnesium, in dantrolene suppression of malignant hyperthermia. Proceedings of the National Academy of Sciences of the United States of America. PMID 28442565 DOI: 10.1073/Pnas.1704103114  0.402
2016 Wu F, Mi W, Fu Y, Struyk A, Cannon SC. Mice with an NaV1.4 sodium channel null allele have latent myasthenia, without susceptibility to periodic paralysis. Brain : a Journal of Neurology. PMID 27048647 DOI: 10.1093/Brain/Aww070  0.833
2016 Cannon SC. When all is lost…a severe myopathy with hypotonia from sodium channel mutations. Brain : a Journal of Neurology. 139: 642-4. PMID 26917582 DOI: 10.1093/Brain/Awv400  0.625
2016 Nelson BR, Makarewich CA, Anderson DM, Winders BR, Troupes CD, Wu F, Reese AL, McAnally JR, Chen X, Kavalali ET, Cannon SC, Houser SR, Bassel-Duby R, Olson EN. A peptide encoded by a transcript annotated as long noncoding RNA enhances SERCA activity in muscle. Science (New York, N.Y.). 351: 271-275. PMID 26816378 DOI: 10.1126/Science.Aad4076  0.707
2015 Cannon SC. Channelopathies of skeletal muscle excitability. Comprehensive Physiology. 5: 761-90. PMID 25880512 DOI: 10.1002/Cphy.C140062  0.577
2014 Mi W, Rybalchenko V, Cannon SC. Disrupted coupling of gating charge displacement to Na+ current activation for DIIS4 mutations in hypokalemic periodic paralysis. The Journal of General Physiology. 144: 137-45. PMID 25024265 DOI: 10.1085/Jgp.201411199  0.617
2014 Trivedi JR, Cannon SC, Griggs RC. Nondystrophic myotonia: challenges and future directions. Experimental Neurology. 253: 28-30. PMID 24361411 DOI: 10.1016/J.Expneurol.2013.12.005  0.549
2013 Ruff RL, Cannon S. New territory opened by periodic paralysis associated with mitochondrial DNA mutation Neurology. 81: 1806-1807. PMID 24153442 DOI: 10.1212/01.Wnl.0000436072.13783.78  0.383
2013 Wu F, Mi W, Cannon SC. Beneficial effects of bumetanide in a CaV1.1-R528H mouse model of hypokalaemic periodic paralysis. Brain : a Journal of Neurology. 136: 3766-74. PMID 24142145 DOI: 10.1093/Brain/Awt280  0.809
2013 Nelson BR, Wu F, Liu Y, Anderson DM, McAnally J, Lin W, Cannon SC, Bassel-Duby R, Olson EN. Skeletal muscle-specific T-tubule protein STAC3 mediates voltage-induced Ca2+ release and contractility. Proceedings of the National Academy of Sciences of the United States of America. 110: 11881-6. PMID 23818578 DOI: 10.1073/Pnas.1310571110  0.731
2013 Trivedi JR, Bundy B, Statland J, Salajegheh M, Rayan DR, Venance SL, Wang Y, Fialho D, Matthews E, Cleland J, Gorham N, Herbelin L, Cannon S, Amato A, Griggs RC, et al. Non-dystrophic myotonia: prospective study of objective and patient reported outcomes. Brain : a Journal of Neurology. 136: 2189-200. PMID 23771340 DOI: 10.1093/Brain/Awt133  0.572
2013 Wu F, Mi W, Cannon SC. Bumetanide prevents transient decreases in muscle force in murine hypokalemic periodic paralysis. Neurology. 80: 1110-6. PMID 23427324 DOI: 10.1212/Wnl.0B013E3182886A0E  0.757
2013 Mi W, Cannon SC. Coupling of Charge Displacement to Na Current Activation is Impaired for DIIS4 Mutations in HypoPP Biophysical Journal. 104: 137a-138a. DOI: 10.1016/J.Bpj.2012.11.784  0.552
2012 Wu F, Mi W, Hernández-Ochoa EO, Burns DK, Fu Y, Gray HF, Struyk AF, Schneider MF, Cannon SC. A calcium channel mutant mouse model of hypokalemic periodic paralysis. The Journal of Clinical Investigation. 122: 4580-91. PMID 23187123 DOI: 10.1172/Jci66091  0.842
2012 Cannon SC. Small fiber neuropathy: a bit less idiopathic? Neurology. 78: 1626-7. PMID 22539571 DOI: 10.1212/Wnl.0B013E318257517A  0.352
2012 Liu Y, Sugiura Y, Wu F, Mi W, Taketo MM, Cannon S, Carroll T, Lin W. β-Catenin stabilization in skeletal muscles, but not in motor neurons, leads to aberrant motor innervation of the muscle during neuromuscular development in mice. Developmental Biology. 366: 255-67. PMID 22537499 DOI: 10.1016/J.Ydbio.2012.04.003  0.68
2011 Wu F, Mi W, Burns DK, Fu Y, Gray HF, Struyk AF, Cannon SC. A sodium channel knockin mutant (NaV1.4-R669H) mouse model of hypokalemic periodic paralysis. The Journal of Clinical Investigation. 121: 4082-94. PMID 21881211 DOI: 10.1172/Jci57398  0.857
2011 Kudryashova E, Struyk A, Mokhonova E, Cannon SC, Spencer MJ. The common missense mutation D489N in TRIM32 causing limb girdle muscular dystrophy 2H leads to loss of the mutated protein in knock-in mice resulting in a Trim32-null phenotype. Human Molecular Genetics. 20: 3925-32. PMID 21775502 DOI: 10.1093/Hmg/Ddr311  0.714
2011 Cheng CJ, Lin SH, Lo YF, Yang SS, Hsu YJ, Cannon SC, Huang CL. Identification and functional characterization of Kir2.6 mutations associated with non-familial hypokalemic periodic paralysis. The Journal of Biological Chemistry. 286: 27425-35. PMID 21665951 DOI: 10.1074/Jbc.M111.249656  0.626
2011 Francis DG, Rybalchenko V, Struyk A, Cannon SC. Leaky sodium channels from voltage sensor mutations in periodic paralysis, but not paramyotonia. Neurology. 76: 1635-41. PMID 21490317 DOI: 10.1212/Wnl.0B013E318219Fb57  0.781
2011 Fu Y, Struyk A, Markin V, Cannon S. Gating behaviour of sodium currents in adult mouse muscle recorded with an improved two-electrode voltage clamp. The Journal of Physiology. 589: 525-46. PMID 21135045 DOI: 10.1113/Jphysiol.2010.199430  0.804
2011 Cheng CJ, Lin SH, Lo YF, Yang SS, Hsu YJ, Cannon SC, Huang CL. Identification and functional characterization of Kir2.6 mutations associated with non-familial hypokalemic periodic paralysis (Journal of Biological Chemistry (2011) 286, (27425-27435)) Journal of Biological Chemistry. 286: 33707. DOI: 10.1074/Jbc.A111.249656  0.384
2011 Hernández-Ochoa EO, Fu V, Mi W, Schneider MF, Cannon SC. Effects of Cav1.1 Mutation (S4-II-R528H) Causing Hypokalemic Periodic Paralysis on L-Type Calcium Current and Voltage Dependent Calcium Release in Isolated Muscle Fibers Biophysical Journal. 100: 591a. DOI: 10.1016/J.Bpj.2010.12.3410  0.481
2010 Cannon SC. Voltage-sensor mutations in channelopathies of skeletal muscle. The Journal of Physiology. 588: 1887-95. PMID 20156847 DOI: 10.1113/Jphysiol.2010.186874  0.615
2010 Cannon SC, Bean BP. Sodium channels gone wild: resurgent current from neuronal and muscle channelopathies. The Journal of Clinical Investigation. 120: 80-3. PMID 20038809 DOI: 10.1172/Jci41340  0.596
2010 Matthews E, Fialho D, Tan SV, Venance SL, Cannon SC, Sternberg D, Fontaine B, Amato AA, Barohn RJ, Griggs RC, Hanna MG. The non-dystrophic myotonias: molecular pathogenesis, diagnosis and treatment. Brain : a Journal of Neurology. 133: 9-22. PMID 19917643 DOI: 10.1093/Brain/Awp294  0.575
2010 Francis D, Struyk A, Cannon SC. Arginine Mutations in the S4 VSD of NaV1.4 Associated with Hypokalemic Periodic Paralysis, But Not with Paramyotonia, Create a Gating Pore Conductance Biophysical Journal. 98: 111a. DOI: 10.1016/J.Bpj.2009.12.4199  0.784
2009 Cannon SC. Getting a charge out of periodic paralysis? Neurology. 72: 1540-1. PMID 19414720 DOI: 10.1212/01.Wnl.0000345878.82061.Cb  0.607
2009 Webb J, Wu FF, Cannon SC. Slow inactivation of the NaV1.4 sodium channel in mammalian cells is impeded by co-expression of the beta1 subunit. Pflã¼Gers Archiv : European Journal of Physiology. 457: 1253-63. PMID 18941776 DOI: 10.1007/S00424-008-0600-8  0.789
2009 Struyk A, Cannon SC. Gating Pore Currents from S4 Mutations of NaV1.4: A Common Pathomechanism in Hypokalemic Periodic Paralysis Biophysical Journal. 96: 203a. DOI: 10.1016/J.Bpj.2008.12.1095  0.769
2009 Cannon SC. Mechanistic Diversity for Channelopathies of Brain and Skeletal Muscle Biophysical Journal. 96: 202a. DOI: 10.1016/J.Bpj.2008.12.1091  0.602
2008 Struyk AF, Markin VS, Francis D, Cannon SC. Gating pore currents in DIIS4 mutations of NaV1.4 associated with periodic paralysis: saturation of ion flux and implications for disease pathogenesis. The Journal of General Physiology. 132: 447-64. PMID 18824591 DOI: 10.1085/Jgp.200809967  0.78
2008 Hayward LJ, Kim JS, Lee MY, Zhou H, Kim JW, Misra K, Salajegheh M, Wu FF, Matsuda C, Reid V, Cros D, Hoffman EP, Renaud JM, Cannon SC, Brown RH. Targeted mutation of mouse skeletal muscle sodium channel produces myotonia and potassium-sensitive weakness. The Journal of Clinical Investigation. 118: 1437-49. PMID 18317596 DOI: 10.1172/Jci32638  0.823
2008 Struyk AF, Cannon SC. Paradoxical depolarization of BA2+- treated muscle exposed to low extracellular K+: insights into resting potential abnormalities in hypokalemic paralysis. Muscle & Nerve. 37: 326-37. PMID 18041053 DOI: 10.1002/Mus.20928  0.718
2008 Webb J, Cannon SC. Cold-induced defects of sodium channel gating in atypical periodic paralysis plus myotonia. Neurology. 70: 755-61. PMID 17898326 DOI: 10.1212/01.Wnl.0000265397.70057.D8  0.73
2007 Struyk AF, Cannon SC. A Na+ channel mutation linked to hypokalemic periodic paralysis exposes a proton-selective gating pore. The Journal of General Physiology. 130: 11-20. PMID 17591984 DOI: 10.1085/Jgp.200709755  0.81
2007 Cannon SC. Physiologic principles underlying ion channelopathies. Neurotherapeutics : the Journal of the American Society For Experimental Neurotherapeutics. 4: 174-83. PMID 17395127 DOI: 10.1016/J.Nurt.2007.01.015  0.495
2006 Cannon SC. Pathomechanisms in channelopathies of skeletal muscle and brain. Annual Review of Neuroscience. 29: 387-415. PMID 16776591 DOI: 10.1146/Annurev.Neuro.29.051605.112815  0.54
2006 Venance SL, Cannon SC, Fialho D, Fontaine B, Hanna MG, Ptacek LJ, Tristani-Firouzi M, Tawil R, Griggs RC. The primary periodic paralyses: diagnosis, pathogenesis and treatment. Brain : a Journal of Neurology. 129: 8-17. PMID 16195244 DOI: 10.1093/Brain/Awh639  0.54
2005 Menard C, Horvitz HR, Cannon S. Chimeric mutations in the M2 segment of the 5-hydroxytryptamine-gated chloride channel MOD-1 define a minimal determinant of anion/cation permeability. The Journal of Biological Chemistry. 280: 27502-7. PMID 15878844 DOI: 10.1074/Jbc.M501624200  0.393
2005 Wu FF, Gordon E, Hoffman EP, Cannon SC. A C-terminal skeletal muscle sodium channel mutation associated with myotonia disrupts fast inactivation. The Journal of Physiology. 565: 371-80. PMID 15774523 DOI: 10.1113/Jphysiol.2005.082909  0.821
2004 Berg J, Jiang H, Thornton CA, Cannon SC. Truncated ClC-1 mRNA in myotonic dystrophy exerts a dominant-negative effect on the Cl current. Neurology. 63: 2371-5. PMID 15623702 DOI: 10.1212/01.Wnl.0000148482.40683.88  0.526
2004 Cannon SC. Paying the price at the pump: dystonia from mutations in a Na+/K+ -ATPase. Neuron. 43: 153-4. PMID 15260948 DOI: 10.1016/J.Neuron.2004.07.002  0.461
2003 Tsujino A, Maertens C, Ohno K, Shen XM, Fukuda T, Harper CM, Cannon SC, Engel AG. Myasthenic syndrome caused by mutation of the SCN4A sodium channel. Proceedings of the National Academy of Sciences of the United States of America. 100: 7377-82. PMID 12766226 DOI: 10.1073/Pnas.1230273100  0.815
2002 Struyk AF, Cannon SC. Slow inactivation does not block the aqueous accessibility to the outer pore of voltage-gated Na channels. The Journal of General Physiology. 120: 509-16. PMID 12356853 DOI: 10.1085/Jgp.20028672  0.721
2002 Mankodi A, Takahashi MP, Jiang H, Beck CL, Bowers WJ, Moxley RT, Cannon SC, Thornton CA. Expanded CUG repeats trigger aberrant splicing of ClC-1 chloride channel pre-mRNA and hyperexcitability of skeletal muscle in myotonic dystrophy. Molecular Cell. 10: 35-44. PMID 12150905 DOI: 10.1016/S1097-2765(02)00563-4  0.653
2002 Cannon SC. An expanding view for the molecular basis of familial periodic paralysis. Neuromuscular Disorders : Nmd. 12: 533-43. PMID 12117476 DOI: 10.1016/S0960-8966(02)00007-X  0.569
2002 Cannon SC. Sodium channel gating: no margin for error. Neuron. 34: 853-4. PMID 12086630 DOI: 10.1016/S0896-6273(02)00735-3  0.583
2001 Takahashi MP, Cannon SC. Mexiletine block of disease-associated mutations in S6 segments of the human skeletal muscle Na(+) channel. The Journal of Physiology. 537: 701-14. PMID 11744749 DOI: 10.1111/J.1469-7793.2001.00701.X  0.587
2001 Wu FF, Takahashi MP, Pegoraro E, Angelini C, Colleselli P, Cannon SC, Hoffman EP. A new mutation in a family with cold-aggravated myotonia disrupts Na(+) channel inactivation. Neurology. 56: 878-84. PMID 11294924 DOI: 10.1212/Wnl.56.7.878  0.585
2001 Cannon SC. Voltage-gated ion channelopathies of the nervous system Clinical Neuroscience Research. 1: 104-117. DOI: 10.1016/S1566-2772(00)00011-6  0.53
2000 Struyk AF, Scoggan KA, Bulman DE, Cannon SC. The human skeletal muscle Na channel mutation R669H associated with hypokalemic periodic paralysis enhances slow inactivation. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 20: 8610-7. PMID 11102465 DOI: 10.1523/Jneurosci.20-23-08610.2000  0.803
2000 Ranganathan R, Cannon SC, Horvitz HR. MOD-1 is a serotonin-gated chloride channel that modulates locomotory behaviour in C. elegans. Nature. 408: 470-5. PMID 11100728 DOI: 10.1038/35044083  0.336
2000 Morrill JA, Cannon SC. COOH-terminal truncated alpha(1S) subunits conduct current better than full-length dihydropyridine receptors. The Journal of General Physiology. 116: 341-8. PMID 10962012 DOI: 10.1085/Jgp.116.3.341  0.622
2000 Ruff RL, Cannon SC. Defective slow inactivation of sodium channels contributes to familial periodic paralysis. Neurology. 54: 2190-2. PMID 10851401  0.353
2000 Vedantham V, Cannon SC. Rapid and slow voltage-dependent conformational changes in segment IVS6 of voltage-gated Na(+) channels. Biophysical Journal. 78: 2943-58. PMID 10827974 DOI: 10.1016/S0006-3495(00)76834-2  0.712
2000 Cannon SC. Spectrum of sodium channel disturbances in the nondystrophic myotonias and periodic paralyses. Kidney International. 57: 772-9. PMID 10720928 DOI: 10.1046/J.1523-1755.2000.00914.X  0.596
2000 Cannon SC. Ion channels and disease Annals of Neurology. 48: 404-404. DOI: 10.1002/1531-8249(200009)48:3<404::Aid-Ana26>3.0.Co;2-5  0.377
1999 Morrill JA, Cannon SC. Effects of mutations causing hypokalaemic periodic paralysis on the skeletal muscle L-type Ca2+ channel expressed in Xenopus laevis oocytes. The Journal of Physiology. 520: 321-36. PMID 10523403 DOI: 10.1111/J.1469-7793.1999.00321.X  0.697
1999 Hayward LJ, Sandoval GM, Cannon SC. Defective slow inactivation of sodium channels contributes to familial periodic paralysis. Neurology. 52: 1447-53. PMID 10227633 DOI: 10.1212/Wnl.52.7.1447  0.715
1999 Takahashi MP, Cannon SC. Enhanced slow inactivation by V445M: a sodium channel mutation associated with myotonia. Biophysical Journal. 76: 861-8. PMID 9929487 DOI: 10.1016/S0006-3495(99)77249-8  0.626
1999 Vedantham V, Cannon SC. The position of the fast-inactivation gate during lidocaine block of voltage-gated Na+ channels. The Journal of General Physiology. 113: 7-16. PMID 9874684 DOI: 10.1085/Jgp.113.1.7  0.722
1998 Morrill JA, Brown RH, Cannon SC. Gating of the L-type Ca channel in human skeletal myotubes: an activation defect caused by the hypokalemic periodic paralysis mutation R528H. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 18: 10320-34. PMID 9852570 DOI: 10.1523/Jneurosci.18-24-10320.1998  0.702
1998 Green DS, George AL, Cannon SC. Human sodium channel gating defects caused by missense mutations in S6 segments associated with myotonia: S804F and V1293I. The Journal of Physiology. 510: 685-94. PMID 9660885 DOI: 10.1111/J.1469-7793.1998.685Bj.X  0.608
1998 Vedantham V, Cannon SC. Slow inactivation does not affect movement of the fast inactivation gate in voltage-gated Na+ channels. The Journal of General Physiology. 111: 83-93. PMID 9417137 DOI: 10.1085/Jgp.111.1.83  0.722
1997 Green DS, Hayward LJ, George AL, Cannon SC. A proposed mutation, Val781Ile, associated with hyperkalemic periodic paralysis and cardiac dysrhythmia is a benign polymorphism. Annals of Neurology. 42: 253-6. PMID 9266738 DOI: 10.1002/Ana.410420219  0.671
1997 Cannon SC. From mutation to myotonia in sodium channel disorders. Neuromuscular Disorders : Nmd. 7: 241-9. PMID 9196906 DOI: 10.1016/S0960-8966(97)00430-6  0.603
1997 Hayward LJ, Brown RH, Cannon SC. Slow inactivation differs among mutant Na channels associated with myotonia and periodic paralysis. Biophysical Journal. 72: 1204-19. PMID 9138567 DOI: 10.1016/S0006-3495(97)78768-X  0.757
1996 Grosson CL, Cannon SC, Corey DP, Gusella JF. Sequence of the voltage-gated sodium channel beta1-subunit in wild-type and in quivering mice. Brain Research. Molecular Brain Research. 42: 222-6. PMID 9013777 DOI: 10.1016/S0169-328X(96)00123-4  0.621
1996 Cannon SC. Sodium channel defects in myotonia and periodic paralysis. Annual Review of Neuroscience. 19: 141-64. PMID 8833439 DOI: 10.1146/Annurev.Ne.19.030196.001041  0.564
1996 Cannon SC. Slow inactivation of sodium channels: more than just a laboratory curiosity. Biophysical Journal. 71: 5-7. PMID 8804584 DOI: 10.1016/S0006-3495(96)79203-2  0.483
1996 Cannon SC. Ion-channel defects and aberrant excitability in myotonia and periodic paralysis. Trends in Neurosciences. 19: 3-10. PMID 8787138 DOI: 10.1016/0166-2236(96)81859-5  0.585
1996 Hayward LJ, Brown RH, Cannon SC. Inactivation defects caused by myotonia-associated mutations in the sodium channel III-IV linker. The Journal of General Physiology. 107: 559-76. PMID 8740371 DOI: 10.1085/Jgp.107.5.559  0.739
1995 Chen C, Cannon SC. Modulation of Na+ channel inactivation by the beta 1 subunit: a deletion analysis. Pflã¼Gers Archiv : European Journal of Physiology. 431: 186-95. PMID 9026778 DOI: 10.1007/Bf00410190  0.433
1995 Cannon SC, Hayward LJ, Beech J, Brown RH. Sodium channel inactivation is impaired in equine hyperkalemic periodic paralysis. Journal of Neurophysiology. 73: 1892-9. PMID 7623088 DOI: 10.1152/Jn.1995.73.5.1892  0.751
1994 Cannon SC. Slow sodium channel inactivation need not be disrupted in the Pathogenesis of myotonia and Periodic Paralysis Biophysical Journal. 66: 543-544. DOI: 10.1016/S0006-3495(94)80808-2  0.494
1993 Cannon SC, Brown RH, Corey DP. Theoretical reconstruction of myotonia and paralysis caused by incomplete inactivation of sodium channels. Biophysical Journal. 65: 270-88. PMID 8396455 DOI: 10.1016/S0006-3495(93)81045-2  0.706
1993 McClatchey AI, Cannon SC, Slaugenhaupt SA, Gusella JF. The cloning and expression of a sodium channel beta 1-subunit cDNA from human brain. Human Molecular Genetics. 2: 745-9. PMID 8394762 DOI: 10.1093/Hmg/2.6.745  0.417
1993 Cannon SC, Strittmatter SM. Functional expression of sodium channel mutations identified in families with periodic paralysis. Neuron. 10: 317-26. PMID 8382500 DOI: 10.1016/0896-6273(93)90321-H  0.569
1993 Cannon SC, Corey DP. Loss of Na+ channel inactivation by anemone toxin (ATX II) mimics the myotonic state in hyperkalaemic periodic paralysis. The Journal of Physiology. 466: 501-20. PMID 8105077 DOI: 10.1113/Jphysiol.1993.Sp019731  0.723
1993 Cannon SC, McClatchey AI, Gusella JF. Modification of the Na+ current conducted by the rat skeletal muscle alpha subunit by coexpression with a human brain beta subunit. Pflã¼Gers Archiv : European Journal of Physiology. 423: 155-7. PMID 7683789 DOI: 10.1007/Bf00374974  0.354
1991 Cannon SC, Brown RH, Corey DP. A sodium channel defect in hyperkalemic periodic paralysis: potassium-induced failure of inactivation. Neuron. 6: 619-26. PMID 1849724 DOI: 10.1016/0896-6273(91)90064-7  0.746
1987 Cannon SC, Robinson DA. Loss of the neural integrator of the oculomotor system from brain stem lesions in monkey. Journal of Neurophysiology. 57: 1383-409. PMID 3585473 DOI: 10.1152/Jn.1987.57.5.1383  0.423
1985 Cannon SC, Robinson DA. An improved neural-network model for the neural integrator of the oculomotor system: more realistic neuron behavior. Biological Cybernetics. 53: 93-108. PMID 4084616 DOI: 10.1007/Bf00337026  0.416
1983 Cannon SC, Robinson DA, Shamma S. A proposed neural network for the integrator of the oculomotor system. Biological Cybernetics. 49: 127-36. PMID 6661444 DOI: 10.1007/Bf00320393  0.422
1982 Cannon SC, Zahalak GI. The mechanical behavior of active human skeletal muscle in small oscillations. Journal of Biomechanics. 15: 111-21. PMID 7076686 DOI: 10.1016/0021-9290(82)90043-4  0.325
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