Cynthia E. Balatoni, Ph.D.

Affiliations: 
2008 University of California, Los Angeles, Los Angeles, CA 
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"Cynthia Balatoni"

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Michael Teitell grad student 2008 UCLA
 (Investigations in TCL1-mediated lymphomagenesis involving polynucleotide phosphorylase and STK39 hypermethylation.)
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Balatoni CE, Dawson DW, Suh J, et al. (2009) Epigenetic silencing of Stk39 in B-cell lymphoma inhibits apoptosis from genotoxic stress. The American Journal of Pathology. 175: 1653-61
Frank MJ, Dawson DW, Bensinger SJ, et al. (2009) Expression of sprouty2 inhibits B-cell proliferation and is epigenetically silenced in mouse and human B-cell lymphomas. Blood. 113: 2478-87
Vaysberg M, Balatoni CE, Nepomuceno RR, et al. (2007) Rapamycin inhibits proliferation of Epstein-Barr virus-positive B-cell lymphomas through modulation of cell-cycle protein expression. Transplantation. 83: 1114-21
French SW, Dawson DW, Chen HW, et al. (2007) The TCL1 oncoprotein binds the RNase PH domains of the PNPase exoribonuclease without affecting its RNA degrading activity. Cancer Letters. 248: 198-210
Chen HW, Rainey RN, Balatoni CE, et al. (2006) Mammalian polynucleotide phosphorylase is an intermembrane space RNase that maintains mitochondrial homeostasis Molecular and Cellular Biology. 26: 8475-8487
Vaysberg M, Balatoni CE, Nepomuceno RR, et al. (2004) RAPAMYCIN INHIBITS EPSTEIN BARR VIRUS+ B CELL LYMPHOMA PROLIFERATION THROUGH MODULATION OF CELL CYCLE PROTEIN EXPRESSION. Transplantation. 78: 76
Nepomuceno RR, Balatoni CE, Natkunam Y, et al. (2003) Rapamycin inhibits the interleukin 10 signal transduction pathway and the growth of Epstein Barr virus B-cell lymphomas. Cancer Research. 63: 4472-80
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