John J. Lemasters
Affiliations: | Biomedical Engineering Joint Program | University of North Carolina, Chapel Hill, Chapel Hill, NC |
Area:
BiomedicalGoogle:
"John Lemasters"Children
Sign in to add traineeSteven P. Elmore | grad student | 2000 | UNC Chapel Hill |
James R. Blattner | grad student | 2005 | UNC Chapel Hill |
Insil Kim | grad student | 2007 | UNC Chapel Hill |
Venkat K. Ramshesh | grad student | 2007 | UNC Chapel Hill |
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Publications
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Hu J, Nieminen AL, Weemhoff JL, et al. (2023) The mitochondrial calcium uniporter mediates mitochondrial Fe uptake and hepatotoxicity after acetaminophen. Toxicology and Applied Pharmacology. 479: 116722 |
Kholmukhamedov A, Li L, Lindsey CC, et al. (2022) A new fluorescent sensor mitoferrofluor indicates the presence of chelatable iron in polarized and depolarized mitochondria. The Journal of Biological Chemistry. 102336 |
Adelusi OB, Ramachandran A, Lemasters JJ, et al. (2022) The role of Iron in lipid peroxidation and protein nitration during acetaminophen-induced liver injury in mice. Toxicology and Applied Pharmacology. 445: 116043 |
Samuvel DJ, Li L, Krishnasamy Y, et al. (2022) Mitochondrial depolarization after acute ethanol treatment drives mitophagy in living mice. Autophagy. 1-15 |
Lemasters JJ. (2021) Metabolic implications of non-Electrogenic ATP/ADP exchange in Cancer cells: A mechanistic basis for the Warburg effect. Biochimica Et Biophysica Acta. Bioenergetics. 148410 |
Wimborne HJ, Hu J, Takemoto K, et al. (2020) Aldehyde dehydrogenase-2 activation decreases acetaminophen hepatotoxicity by prevention of mitochondrial depolarization. Toxicology and Applied Pharmacology. 114982 |
Hu J, Lemasters JJ. (2020) Suppression of iron mobilization from lysosomes to mitochondria attenuates liver injury after acetaminophen overdose in vivo in mice: Protection by minocycline. Toxicology and Applied Pharmacology. 114930 |
Lemasters JJ, Jaeschke H. (2020) Oxidative Stress and Inflammation in the Liver Liver. 714-727 |
Krishnasamy Y, Gooz M, Li L, et al. (2019) Role of mitochondrial depolarization and disrupted mitochondrial homeostasis in non-alcoholic steatohepatitis and fibrosis in mice. International Journal of Physiology, Pathophysiology and Pharmacology. 11: 190-204 |
Heslop KA, Rovini A, Hunt EG, et al. (2019) JNK activation and translocation to mitochondria mediates mitochondrial dysfunction and cell death induced by VDAC opening and sorafenib in hepatocarcinoma cells. Biochemical Pharmacology. 113728 |