Zhongsheng You, Ph.D.
Affiliations: | 2002 | University of California, San Diego, La Jolla, CA |
Area:
Cell CycleGoogle:
"Zhongsheng You"Parents
Sign in to add mentorJohn Newport | grad student | 2002 | UCSD | |
(Sensing and responding to DNA replication.) |
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Publications
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Zhao H, Li J, You Z, et al. (2024) Distinct regulation of ATM signaling by DNA single-strand breaks and APE1. Nature Communications. 15: 6517 |
Kong L, Cheng C, Cheruiyot A, et al. (2024) TCAF1 promotes TRPV2-mediated Ca release in response to cytosolic DNA to protect stressed replication forks. Nature Communications. 15: 4609 |
Jaiswal RK, Lei KH, Chastain M, et al. (2023) CaMKK2 and CHK1 phosphorylate human STN1 in response to replication stress to protect stalled forks from aberrant resection. Nature Communications. 14: 7882 |
Li Y, Wang F, Li X, et al. (2023) The ATM-E6AP-MASTL axis mediates DNA damage checkpoint recovery. Elife. 12 |
Li Y, Wang F, Li X, et al. (2023) The ATM-E6AP-MASTL axis mediates DNA damage checkpoint recovery. Biorxiv : the Preprint Server For Biology |
Yang Z, Lemacon DS, Li S, et al. (2022) Context-dependent pro- and anti-resection roles of ZKSCAN3 in the regulation of fork processing during replication stress. The Journal of Biological Chemistry. 102215 |
Cheruiyot A, Li S, Nonavinkere Srivatsan S, et al. (2021) Nonsense-mediated RNA Decay Is a Unique Vulnerability of Cancer Cells harboring SF3B1 or U2AF1 mutations. Cancer Research |
Zhu C, Rogers A, Asleh K, et al. (2020) Phospho-Ser-VCP Is Required for DNA Damage Response and Is Associated with Poor Prognosis of Chemotherapy-Treated Breast Cancer. Cell Reports. 31: 107745 |
Li S, Lavagnino Z, Lemacon D, et al. (2019) Ca-Stimulated AMPK-Dependent Phosphorylation of Exo1 Protects Stressed Replication Forks from Aberrant Resection. Molecular Cell |
Zhao XT, You ZS, Cheng P, et al. (2019) Investigation of Protein-DNA Interactions in Enhancer II and Core Promoter of HBV by in vivo Footprinting. Sheng Wu Hua Xue Yu Sheng Wu Wu Li Xue Bao Acta Biochimica Et Biophysica Sinica. 31: 489-493 |