Sarah A. Stanley
Affiliations: | Molecular and Cell Biology | University of California, Berkeley, Berkeley, CA, United States |
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"Sarah Stanley"
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| Knight M, Stanley S. (2019) HIF-1α as a central mediator of cellular resistance to intracellular pathogens. Current Opinion in Immunology. 60: 111-116 |
| Van Dis E, Sogi KM, Rae CS, et al. (2018) STING-Activating Adjuvants Elicit a Th17 Immune Response and Protect against Mycobacterium tuberculosis Infection. Cell Reports. 23: 1435-1447 |
| Knight M, Braverman J, Asfaha K, et al. (2018) Lipid droplet formation in Mycobacterium tuberculosis infected macrophages requires IFN-γ/HIF-1α signaling and supports host defense. Plos Pathogens. 14: e1006874 |
| Braverman J, Stanley SA. (2017) Nitric Oxide Modulates Macrophage Responses to Mycobacterium tuberculosis Infection through Activation of HIF-1α and Repression of NF-κB. Journal of Immunology (Baltimore, Md. : 1950) |
| Sogi KM, Lien KA, Johnson J, et al. (2017) The tyrosine kinase inhibitor gefitinib restricts Mycobacterium tuberculosis growth through increased lysosomal biogenesis and modulation of cytokine signaling. Acs Infectious Diseases |
| Braverman J, Sogi KM, Benjamin D, et al. (2016) HIF-1α Is an Essential Mediator of IFN-γ-Dependent Immunity to Mycobacterium tuberculosis. Journal of Immunology (Baltimore, Md. : 1950) |
| Stanley SA, Barczak AK, Silvis MR, et al. (2014) Identification of host-targeted small molecules that restrict intracellular Mycobacterium tuberculosis growth. Plos Pathogens. 10: e1003946 |
| Grant SS, Kawate T, Nag PP, et al. (2013) Identification of novel inhibitors of nonreplicating Mycobacterium tuberculosis using a carbon starvation model. Acs Chemical Biology. 8: 2224-34 |
| Stanley SA, Cox JS. (2013) Host-pathogen interactions during Mycobacterium tuberculosis infections. Current Topics in Microbiology and Immunology. 374: 211-41 |
| Stanley SA, Kawate T, Iwase N, et al. (2013) Diarylcoumarins inhibit mycolic acid biosynthesis and kill Mycobacterium tuberculosis by targeting FadD32. Proceedings of the National Academy of Sciences of the United States of America. 110: 11565-70 |