Maureen Harrington

Biochemistry and Molecular Biology Indiana University, Bloomington, Bloomington, IN, United States 
Molecular Biology
"Maureen Harrington"
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Zhao W, Breese E, Bowers A, et al. (2013) SIMPL enhancement of tumor necrosis factor-α dependent p65-MED1 complex formation is required for mammalian hematopoietic stem and progenitor cell function. Plos One. 8: e61123
Lin L, Wagner MC, Cocklin R, et al. (2011) The antibiotic gentamicin inhibits specific protein trafficking functions of the Arf1/2 family of GTPases. Antimicrobial Agents and Chemotherapy. 55: 246-54
Laing BM, Barrow-Laing L, Harrington M, et al. (2010) Properties of double-stranded oligonucleotides modified with lipophilic substituents. Bioconjugate Chemistry. 21: 1537-44
Benson EA, Goebl MG, Yang FC, et al. (2010) Loss of SIMPL compromises TNF-alpha-dependent survival of hematopoietic progenitors. Experimental Hematology. 38: 71-81
Lockett A, Goebl MG, Harrington MA. (2008) Transient membrane recruitment of IRAK-1 in response to LPS and IL-1beta requires TNF R1. American Journal of Physiology. Cell Physiology. 295: C313-23
Ott LW, Resing KA, Sizemore AW, et al. (2007) Tumor Necrosis Factor-alpha- and interleukin-1-induced cellular responses: coupling proteomic and genomic information. Journal of Proteome Research. 6: 2176-85
Luo Y, Kwon HJ, Montano S, et al. (2007) Phosphorylation of SIMPL modulates RelA-associated NF-kappaB-dependent transcription. American Journal of Physiology. Cell Physiology. 292: C1013-23
Haag Breese E, Uversky VN, Georgiadis MM, et al. (2006) The disordered amino-terminus of SIMPL interacts with members of the 70-kDa heat-shock protein family. Dna and Cell Biology. 25: 704-14
Vig-Varga E, Benson EA, Limbil TL, et al. (2006) Alpha-lipoic acid modulates ovarian surface epithelial cell growth. Gynecologic Oncology. 103: 45-52
Kim JA, Yeh DC, Ver M, et al. (2005) Phosphorylation of Ser24 in the pleckstrin homology domain of insulin receptor substrate-1 by Mouse Pelle-like kinase/interleukin-1 receptor-associated kinase: cross-talk between inflammatory signaling and insulin signaling that may contribute to insulin resistance. The Journal of Biological Chemistry. 280: 23173-83
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