Alec W. Gross, Ph.D.
Affiliations: | 2002 | Brandeis University, Waltham, MA, United States |
Area:
Oncology, Cell Biology, GeneticsGoogle:
"Alec Gross"Parents
Sign in to add mentor| Ruibao Ren | grad student | 2002 | Brandeis | |
| (Dissecting molecular mechanisms of leukemogenesis with a mouse chronic myelogenous leukemia model.) | ||||
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Publications
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| Lu X, Gross AW, Lodish HF. (2006) Active conformation of the erythropoietin receptor: random and cysteine-scanning mutagenesis of the extracellular juxtamembrane and transmembrane domains. The Journal of Biological Chemistry. 281: 7002-11 |
| Ji Y, Gross AW, Zhang X, et al. (2004) The Abl Carboxyl Terminal Region Is Required for the Induction of Acute Lymphoblastic Leukemia by V-Abl but Not for the Induction of Myeloproliferative Disease by BCR/ABL or V-Abl. Blood. 104: 2969-2969 |
| Dinulescu DM, Wood LJ, Shen L, et al. (2003) c-CBL is not required for leukemia induction by Bcr-Abl in mice. Oncogene. 22: 8852-60 |
| Zhang X, Subrahmanyam R, Wong R, et al. (2001) The NH(2)-terminal coiled-coil domain and tyrosine 177 play important roles in induction of a myeloproliferative disease in mice by Bcr-Abl. Molecular and Cellular Biology. 21: 840-53 |
| Gross AW, Ren R. (2000) Bcr-Abl has a greater intrinsic capacity than v-Abl to induce the neoplastic expansion of myeloid cells. Oncogene. 19: 6286-96 |
| Gross AW, Zhang X, Ren R. (1999) Bcr-Abl with an SH3 deletion retains the ability To induce a myeloproliferative disease in mice, yet c-Abl activated by an SH3 deletion induces only lymphoid malignancy. Molecular and Cellular Biology. 19: 6918-28 |