Robert P. Kimberly
Affiliations: | Cell Biology | University of Alabama, Birmingham, Birmingham, AL, United States |
Area:
Cell Biology, ImmunologyGoogle:
"Robert Kimberly"Children
Sign in to add trainee| Xinrui Li | grad student | 2009 | UAB |
| Travis S. Ptacek | grad student | 2013 | UAB |
| Spandan Shah | grad student | 2013 | UAB |
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Publications
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| Zhang Y, Li X, Gibson A, et al. (2020) Skewed allelic expression on X-chromosome associated with aberrant expression of XIST on systemic lupus erythematosus lymphocytes. Human Molecular Genetics |
| Kamitaki N, Sekar A, Handsaker RE, et al. (2020) Complement genes contribute sex-biased vulnerability in diverse disorders. Nature |
| Breitbach M, Ramaker RC, Roberts K, et al. (2019) Epigenetic Defects in the B cell lineage of Systemic Lupus Erythematosus Patients Display Population-Specific Patterns. Arthritis & Rheumatology (Hoboken, N.J.) |
| Zumaquero E, Stone SL, Scharer CD, et al. (2019) IFNγ induces epigenetic programming of human T-bet B cells and promotesTLR7/8 and IL-21 induced differentiation. Elife. 8 |
| Spiliopoulou A, Colombo M, Plant D, et al. (2019) Association of response to TNF inhibitors in rheumatoid arthritis with quantitative trait loci for and CD39. Annals of the Rheumatic Diseases |
| Zumaquero E, Stone SL, Scharer CD, et al. (2019) Author response: IFNγ induces epigenetic programming of human T-bethi B cells and promotes TLR7/8 and IL-21 induced differentiation Elife |
| Laufer VA, Tiwari HK, Reynolds RJ, et al. (2018) Genetic Influences on Susceptibility to Rheumatoid Arthritis in African-Americans. Human Molecular Genetics |
| Martínez-Bueno M, Oparina N, Dozmorov MG, et al. (2018) Trans-Ethnic Mapping of Identifies Two Independent SLE-Risk Linkage Groups Enriched for Co-Transcriptional Splicing Marks. International Journal of Molecular Sciences. 19 |
| Hanscombe KB, Morris DL, Noble JA, et al. (2018) Genetic fine mapping of Systemic Lupus Erythematosus MHC associations in Europeans and African Americans. Human Molecular Genetics |
| Patel Z, Lu X, Miller D, et al. (2018) A plausibly causal functional lupus-associated risk variant in the STAT1-STAT4 locus. Human Molecular Genetics |