Archana Sanjay

Cell Biology Temple University, Philadelphia, PA, United States 
Cell Biology, Molecular Biology
"Archana Sanjay"
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Yu J, Adapala NS, Doherty L, et al. (2019) Cbl-PI3K interaction regulates Cathepsin K secretion in osteoclasts. Bone
Jastrzebski S, Kalinowski J, Mun S, et al. (2019) Protease-Activated Receptor 1 Deletion Causes Enhanced Osteoclastogenesis in Response to Inflammatory Signals through a Notch2-Dependent Mechanism. Journal of Immunology (Baltimore, Md. : 1950)
Adapala NS, Root S, Lorenzo J, et al. (2019) PI3K activation increases SDF-1 production and number of osteoclast precursors, and enhances SDF-1-mediated osteoclast precursor migration. Bone Reports. 10: 100203
Scanlon V, Soung do Y, Adapala NS, et al. (2015) Role of Cbl-PI3K Interaction during Skeletal Remodeling in a Murine Model of Bone Repair. Plos One. 10: e0138194
Adapala NS, Holland D, Scanlon V, et al. (2014) Loss of Cbl-PI3K interaction in mice prevents significant bone loss following ovariectomy. Bone. 67: 1-9
Guzzo RM, Scanlon V, Sanjay A, et al. (2014) Establishment of human cell type-specific iPS cells with enhanced chondrogenic potential. Stem Cell Reviews. 10: 820-9
Jacome-Galarza C, Soung do Y, Adapala NS, et al. (2014) Altered hematopoietic stem cell and osteoclast precursor frequency in cathepsin K null mice. Journal of Cellular Biochemistry. 115: 1449-57
Rafiq K, Kolpakov MA, Seqqat R, et al. (2014) c-Cbl inhibition improves cardiac function and survival in response to myocardial ischemia. Circulation. 129: 2031-43
Adapala NS, Barbe MF, Tsygankov AY, et al. (2014) Loss of Cbl-PI3K interaction enhances osteoclast survival due to p21-Ras mediated PI3K activation independent of Cbl-b. Journal of Cellular Biochemistry. 115: 1277-89
Back SH, Adapala NS, Barbe MF, et al. (2013) TULA-2, a novel histidine phosphatase, regulates bone remodeling by modulating osteoclast function. Cellular and Molecular Life Sciences : Cmls. 70: 1269-84
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