Guruprasad R. Pattar, Ph.D.

Affiliations: 
2014 Cellular and Integrative Physiology Indiana University, Bloomington, Bloomington, IN, United States 
Area:
Physiology Biology, Cell Biology, Molecular Biology
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"Guruprasad Pattar"

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Jeffrey S. Elmendorf grad student 2014 Indiana University
 (A nutrient network regulating cellular cholesterol and glucose metabolism.)
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Publications

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Arthur SN, Cantor LB, WuDunn D, et al. (2014) Efficacy, safety, and survival rates of IOP-lowering effect of phacoemulsification alone or combined with canaloplasty in glaucoma patients. Journal of Glaucoma. 23: 316-20
Bhonagiri P, Pattar GR, Habegger KM, et al. (2011) Evidence coupling increased hexosamine biosynthesis pathway activity to membrane cholesterol toxicity and cortical filamentous actin derangement contributing to cellular insulin resistance. Endocrinology. 152: 3373-84
Bhonagiri P, Pattar GR, Horvath EM, et al. (2010) Hexosamine biosynthesis pathway flux contributes to insulin resistance via altering membrane phosphatidylinositol 4,5-bisphosphate and cortical filamentous actin (Endocrinology (2009) 150, (1636-1645) DOI: 10.1210/en.2008-1102) Endocrinology. 151: 2967
Bhonagiri P, Pattar GR, Horvath EM, et al. (2009) Hexosamine biosynthesis pathway flux contributes to insulin resistance via altering membrane phosphatidylinositol 4,5-bisphosphate and cortical filamentous actin. Endocrinology. 150: 1636-45
Pattar GR, Tackett L, Liu P, et al. (2006) Chromium picolinate positively influences the glucose transporter system via affecting cholesterol homeostasis in adipocytes cultured under hyperglycemic diabetic conditions. Mutation Research. 610: 93-100
Chen G, Liu P, Pattar GR, et al. (2006) Chromium activates glucose transporter 4 trafficking and enhances insulin-stimulated glucose transport in 3T3-L1 adipocytes via a cholesterol-dependent mechanism. Molecular Endocrinology (Baltimore, Md.). 20: 857-70
Chen G, Raman P, Bhonagiri P, et al. (2004) Protective effect of phosphatidylinositol 4,5-bisphosphate against cortical filamentous actin loss and insulin resistance induced by sustained exposure of 3T3-L1 adipocytes to insulin. The Journal of Biological Chemistry. 279: 39705-9
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