Kevin McIver

Cell Biology & Molecular Genetics University of Maryland, College Park, College Park, MD 
Microbiology Biology, Molecular Biology
"Kevin McIver"
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Akhter F, Womack E, Vidal JE, et al. (2021) Hemoglobin induces early and robust biofilm development in by a pathway that involves but not the cognate two-component system. Infection and Immunity
Akhter F, Womack E, Vidal JE, et al. (2020) Hemoglobin stimulates vigorous growth of Streptococcus pneumoniae and shapes the pathogen's global transcriptome. Scientific Reports. 10: 15202
Valdes KM, Sundar GS, Vega LA, et al. (2016) The fruRBA operon is necessary for Group A Streptococcal growth in fructose and for resistance to neutrophil killing during growth in whole human blood. Infection and Immunity
van der Beek SL, Le Breton Y, Ferenbach AT, et al. (2015) GacA is Essential for Group A Streptococcus and Defines a New Class of Monomeric dTDP-4-dehydrorhamnose Reductases (RmlD). Molecular Microbiology
Le Breton Y, Belew AT, Valdes KM, et al. (2015) Essential Genes in the Core Genome of the Human Pathogen Streptococcus pyogenes. Scientific Reports. 5: 9838
Sachla AJ, Le Breton Y, Akhter F, et al. (2014) The crimson conundrum: heme toxicity and tolerance in GAS. Frontiers in Cellular and Infection Microbiology. 4: 159
Freiberg JA, McIver KS, Shirtliff ME. (2014) In vivo expression of Streptococcus pyogenes immunogenic proteins during tibial foreign body infection. Infection and Immunity. 82: 3891-9
Baruch M, Belotserkovsky I, Hertzog BB, et al. (2014) An extracellular bacterial pathogen modulates host metabolism to regulate its own sensing and proliferation. Cell. 156: 97-108
Gera K, Le T, Jamin R, et al. (2014) The phosphoenolpyruvate phosphotransferase system in group A Streptococcus acts to reduce streptolysin S activity and lesion severity during soft tissue infection. Infection and Immunity. 82: 1192-204
Le Breton Y, McIver KS. (2013) Genetic manipulation of Streptococcus pyogenes (the Group A Streptococcus, GAS). Current Protocols in Microbiology. 30: Unit 9D.3.
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