Gabor Kokeny, Ph.D.

Affiliations: 
2007 Semmelweis Egyetem, Hungary 
Area:
Pathology, Molecular Biology
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"Gabor Kokeny"

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Laszlo Rosivall grad student 2007 Semmelweis Egyetem
 (Molecular mechanisms of chronic experimental inflammatory renal diseases.)
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Publications

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Kökény G, Németh Á, Kopp JB, et al. (2022) Susceptibility to kidney fibrosis in mice is associated with early growth response-2 protein and tissue inhibitor of metalloproteinase-1 expression. Kidney International
Popovics H, Mikone K, Mozes M, et al. (2020) P0721Ppar-Gamma Activation Inhibits Tgf-Beta Induced Renal Complement And Galectin-3 Expression In Vivo And In Vitro Nephrology Dialysis Transplantation. 35
Kokeny G, Popovics H, Mikone K, et al. (2020) P0681Timp-1 Deficiency Dampens Renal Galectin-3 Response In Diabetes Nephrology Dialysis Transplantation. 35
Kökény G, Calvier L, Legchenko E, et al. (2019) PPARγ is a gatekeeper for extracellular matrix and vascular cell homeostasis: beneficial role in pulmonary hypertension and renal/cardiac/pulmonary fibrosis. Current Opinion in Nephrology and Hypertension
Németh Á, Mózes MM, Calvier L, et al. (2019) The PPARγ agonist pioglitazone prevents TGF-β induced renal fibrosis by repressing EGR-1 and STAT3. Bmc Nephrology. 20: 245
Nemeth A, Tasnady K, Mozes M, et al. (2019) FP345TIMP-1 DEFICIENCY REDUCES TGF-BETA EXPRESSION AND DAMPENS RENAL FIBROSIS AFTER RENAL ABLATION Nephrology Dialysis Transplantation. 34
Nemeth A, Tasnady K, Mozes M, et al. (2019) SP304PPARγ AGONIST PREVENTS RENAL FIBROSIS BY REPRESSING THE ACTIVATION OF STAT3 AND TIMP-1 Nephrology Dialysis Transplantation. 34
Mozes M, Nemeth A, Fazekas K, et al. (2018) SP270GENETIC SUSCEPTIBILITY TO TGF-BETA INDUCED RENAL FIBROSIS IS ASSOCIATED WITH ALTERED MIR-199 EXPRESSION Nephrology Dialysis Transplantation. 33: i434-i434
Kokeny G, Foss A, Nemeth A, et al. (2018) SP257EARLY RENAL INFLAMMATORY MOLECULE EXPRESSION DIFFERENCES AFTER UNILATERAL URETER OBSTRUCTION IN MICE Nephrology Dialysis Transplantation. 33: i430-i431
Czirok S, Fang L, Radovits T, et al. (2017) Cinaciguat ameliorates glomerular damage by reducing ERK1/2 activity and TGF-ß expression in type-1 diabetic rats. Scientific Reports. 7: 11218
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