Chen-Lin Hsieh, Ph.D.

Affiliations: 
2010 Biology (Cell-Molecular Biology) The University of Toledo 
Area:
Molecular Biology
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"Chen-Lin Hsieh"

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Lirim Shemshedini grad student 2010 The University of Toledo
 (The pro-cancer function of soluble guanylate cyclase alpha-1 (sGCalpha1) in prostate cancer progression.)
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Publications

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Gui B, Hsieh CL, Kantoff PW, et al. (2017) Androgen receptor-mediated downregulation of microRNA-221 and -222 in castration-resistant prostate cancer. Plos One. 12: e0184166
Zhou J, Gao S, Hsieh CL, et al. (2017) Peptide B targets soluble guanylyl cyclase α1 and kills prostate cancer cells. Plos One. 12: e0184088
Hsieh CL, Botta G, Gao S, et al. (2015) PLZF, a Tumor Suppressor Genetically Lost in Metastatic Castration-Resistant Prostate Cancer, Is a Mediator of Resistance to Androgen Deprivation Therapy. Cancer Research. 75: 1944-8
Hsieh CL, Fei T, Chen Y, et al. (2014) Enhancer RNAs participate in androgen receptor-driven looping that selectively enhances gene activation. Proceedings of the National Academy of Sciences of the United States of America. 111: 7319-24
Gao S, Hsieh CL, Zhou J, et al. (2013) Zinc Finger 280B regulates sGCα1 and p53 in prostate cancer cells. Plos One. 8: e78766
Gao S, Hsieh CL, Bhansali M, et al. (2013) A peptide against soluble guanylyl cyclase α1: a new approach to treating prostate cancer. Plos One. 8: e64189
Nguyen HC, Xie W, Yang M, et al. (2013) Expression differences of circulating microRNAs in metastatic castration resistant prostate cancer and low-risk, localized prostate cancer. The Prostate. 73: 346-54
Cai C, Hsieh CL, Gao S, et al. (2012) Soluble guanylyl cyclase α1 and p53 cytoplasmic sequestration and down-regulation in prostate cancer. Molecular Endocrinology (Baltimore, Md.). 26: 292-307
Sun T, Yang M, Chen S, et al. (2012) The altered expression of MiR-221/-222 and MiR-23b/-27b is associated with the development of human castration resistant prostate cancer. The Prostate. 72: 1093-103
Hsieh CL, Cai C, Giwa A, et al. (2008) Expression of a hyperactive androgen receptor leads to androgen-independent growth of prostate cancer cells. Journal of Molecular Endocrinology. 41: 13-23
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