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Daniel E. Koshland grad student 1979 UC Berkeley (Chemistry Tree)
 (Bacterial chemotaxis: genetics and biochemistry of the che genes of Salmonella typhimurium)
William Erwin Paul post-doc 1983 National Institute of Health
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Publications

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Proekt I, Miller CN, Jeanne M, et al. (2016) LYN- and AIRE-mediated tolerance checkpoint defects synergize to trigger organ-specific autoimmunity. The Journal of Clinical Investigation
Beck-Engeser GB, Winkelmann R, Wheeler ML, et al. (2015) APOBEC3 enzymes restrict marginal zone B cells. European Journal of Immunology. 45: 695-704
Rookhuizen DC, DeFranco AL. (2014) Toll-like receptor 9 signaling acts on multiple elements of the germinal center to enhance antibody responses. Proceedings of the National Academy of Sciences of the United States of America. 111: E3224-33
Yu M, Zhou H, Zhao J, et al. (2014) MyD88-dependent interplay between myeloid and endothelial cells in the initiation and progression of obesity-associated inflammatory diseases. The Journal of Experimental Medicine. 211: 887-907
Hua Z, Gross AJ, Lamagna C, et al. (2014) Requirement for MyD88 signaling in B cells and dendritic cells for germinal center anti-nuclear antibody production in Lyn-deficient mice. Journal of Immunology (Baltimore, Md. : 1950). 192: 875-85
Lamagna C, Hu Y, DeFranco AL, et al. (2014) B cell-specific loss of Lyn kinase leads to autoimmunity. Journal of Immunology (Baltimore, Md. : 1950). 192: 919-28
Yu M, Zhou H, Zhao J, et al. (2014) Correction to MyD88-dependent interplay between myeloid and endothelial cells in the initiation and progression of obesity-associated inflammatory diseases [J. Exp. Med., 211, 5, (2014) 887-907] Journal of Experimental Medicine. 211: 1003
Lamagna C, Scapini P, van Ziffle JA, et al. (2013) Hyperactivated MyD88 signaling in dendritic cells, through specific deletion of Lyn kinase, causes severe autoimmunity and inflammation. Proceedings of the National Academy of Sciences of the United States of America. 110: E3311-20
Han D, Walsh MC, Cejas PJ, et al. (2013) Dendritic cell expression of the signaling molecule TRAF6 is critical for gut microbiota-dependent immune tolerance. Immunity. 38: 1211-22
Callahan JA, Hammer GE, Agelides A, et al. (2013) Cutting edge: ABIN-1 protects against psoriasis by restricting MyD88 signals in dendritic cells. Journal of Immunology (Baltimore, Md. : 1950). 191: 535-9
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