Christy L. Trejo, Ph.D.
Affiliations: | 2012 | Biochemistry and Molecular Biology | University of California, San Francisco, San Francisco, CA |
Area:
Molecular Biology, Cell Biology, Biochemistry, OncologyGoogle:
"Christy Trejo"Parents
Sign in to add mentor| Martin McMahon | grad student | 2012 | UCSF | |
| (Analysis of the Initiation, Progression, and Therapy of BRAF(V600E) and KRAS(G12D) - driven Non-Small Cell Lung Adenocarcinoma Using Conditional Mouse Models.) | ||||
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Publications
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| van Veen JE, Scherzer M, Boshuizen J, et al. (2019) Mutationally-activated PI3'-kinase-α promotes de-differentiation of lung tumors initiated by the BRAF oncoprotein kinase. Elife. 8 |
| Veen JEv, Scherzer M, Boshuizen J, et al. (2019) Author response: Mutationally-activated PI3’-kinase-α promotes de-differentiation of lung tumors initiated by the BRAFV600E oncoprotein kinase Elife |
| Green S, Trejo CL, McMahon M. (2015) PIK3CAH1047R Accelerates and Enhances KRASG12D-Driven Lung Tumorigenesis. Cancer Research. 75: 5378-91 |
| Trejo CL, Green S, Marsh V, et al. (2013) Mutationally activated PIK3CA(H1047R) cooperates with BRAF(V600E) to promote lung cancer progression. Cancer Research. 73: 6448-61 |
| Collisson EA, Trejo CL, Silva JM, et al. (2012) A central role for RAF→MEK→ERK signaling in the genesis of pancreatic ductal adenocarcinoma. Cancer Discovery. 2: 685-93 |
| Trejo CL, Juan J, Vicent S, et al. (2012) MEK1/2 inhibition elicits regression of autochthonous lung tumors induced by KRASG12D or BRAFV600E. Cancer Research. 72: 3048-59 |