Kaijie He, Ph.D.

Affiliations: 
2012 Molecular Biology University of Southern California, Los Angeles, CA, United States 
Area:
Molecular Biology, Cell Biology, Oncology
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"Kaijie He"

Parents

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Amir Goldkorn grad student 2012 USC
 (The cancer stem-like phenotype: Therapeutics, phenotypic plasticity and mechanistic studies.)
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Publications

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He K, You H, Li Y, et al. (2016) TCRγ4δ1-engineered αβT cells exhibit effective antitumor activity. Molecular Medicine (Cambridge, Mass.). 22
He K, Qu H, Xu LN, et al. (2016) Epigenetics changes caused by the fusion of human embryonic stem cell and ovarian cancer cells. Bioscience Reports
He K, Chen D, Ruan H, et al. (2016) BRAFV600E-dependent Mcl-1 stabilization leads to everolimus resistance in colon cancer cells. Oncotarget
Chen Y, Jiang T, Shi L, et al. (2016) hcrcn81 promotes cell proliferation through Wnt signaling pathway in colorectal cancer. Medical Oncology (Northwood, London, England). 33: 3
He K, Xu T, Xu Y, et al. (2014) Cancer cells acquire a drug resistant, highly tumorigenic, cancer stem-like phenotype through modulation of the PI3K/Akt/β-catenin/CBP pathway. International Journal of Cancer. Journal International Du Cancer. 134: 43-54
He K, Xu T, Xu Y, et al. (2012) Abstract 3320: Plasticity between the cancer stem-like and non cancer stem-like states is regulated by the PI3K/Akt/β-catenin/CBP pathway Cancer Research. 72: 3320-3320
Xu Y, He K, Goldkorn A. (2011) Telomerase targeted therapy in cancer and cancer stem cells. Clinical Advances in Hematology & Oncology : H&O. 9: 442-55
He K, Xu T, Goldkorn A. (2011) Cancer cells cyclically lose and regain drug-resistant highly tumorigenic features characteristic of a cancer stem-like phenotype. Molecular Cancer Therapeutics. 10: 938-48
Xu T, He K, Wang L, et al. (2011) Prostate tumor cells with cancer progenitor properties have high telomerase activity and are rapidly killed by telomerase interference. The Prostate. 71: 1390-400
He K, Xu T, Goldkorn A. (2010) Abstract 5152: Cancer stem cells arise directly from differentiated cancer cells: A new model of dynamic equilibrium modulated by the Akt pathway Cancer Research. 70: 5152-5152
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