Susan M. Farabaugh, Ph.D.
Affiliations: | 2011 | University of Colorado Anschutz Medical Campus, Denver, Aurora, CO |
Area:
Biochemistry, Molecular BiologyGoogle:
"Susan Farabaugh"Parents
Sign in to add mentor| Heide L. Ford | grad student | 2011 | University of Colorado, Denver | |
| (The role of the Eya2 coactivator in Six1-induced tumor progression.) | ||||
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Publications
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| Farabaugh SM, Litzenburger BC, Elangovan A, et al. (2020) IGF1R constitutive activation expands luminal progenitors and influences lineage differentiation during breast tumorigenesis. Developmental Biology |
| Farabaugh SM, Chan BT, Cui X, et al. (2016) Lack of interaction between ErbB2 and insulin receptor substrate signaling in breast cancer. Cell Communication and Signaling : Ccs. 14: 25 |
| Farabaugh SM, Boone DN, Lee AV. (2015) Role of IGF1R in Breast Cancer Subtypes, Stemness, and Lineage Differentiation. Frontiers in Endocrinology. 6: 59 |
| Iwanaga R, Wang CA, Micalizzi DS, et al. (2012) Expression of Six1 in luminal breast cancers predicts poor prognosis and promotes increases in tumor initiating cells by activation of extracellular signal-regulated kinase and transforming growth factor-beta signaling pathways. Breast Cancer Research : Bcr. 14: R100 |
| Farabaugh SM, Micalizzi DS, Jedlicka P, et al. (2012) Eya2 is required to mediate the pro-metastatic functions of Six1 via the induction of TGF-β signaling, epithelial-mesenchymal transition, and cancer stem cell properties. Oncogene. 31: 552-62 |
| Fini MA, Monks J, Farabaugh SM, et al. (2011) Contribution of xanthine oxidoreductase to mammary epithelial and breast cancer cell differentiation in part modulates inhibitor of differentiation-1. Molecular Cancer Research : McR. 9: 1242-54 |
| Micalizzi DS, Wang CA, Farabaugh SM, et al. (2010) Homeoprotein Six1 increases TGF-beta type I receptor and converts TGF-beta signaling from suppressive to supportive for tumor growth. Cancer Research. 70: 10371-80 |
| Micalizzi DS, Farabaugh SM, Ford HL. (2010) Epithelial-mesenchymal transition in cancer: parallels between normal development and tumor progression. Journal of Mammary Gland Biology and Neoplasia. 15: 117-34 |
| Farabaugh SM, Welm AL, Ford HL. (2009) Abstract A40: Six1 is dependent on its coactivator Eya2 to mediate tumor progression in the MCF7 mammary carcinoma cell line Cancer Research. 69 |