Guy Sauvageau
Affiliations: | Molecular Biology | Université de Montréal, Montréal, Canada |
Area:
Molecular Biology, Cell BiologyGoogle:
"Guy Sauvageau"Children
Sign in to add trainee| Julie Lessard | grad student | 2003 | Université de Montréal |
| Martin Sauvageau | grad student | 2003-2011 | Université de Montréal |
| Sonia Cellot | grad student | 2012 | Université de Montréal |
| Eric Deneault | grad student | 2013 | Université de Montréal |
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Publications
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| Chagraoui J, Girard S, Mallinger L, et al. (2024) KBTBD4-mediated Reduction of MYC Is Critical For Hematopoietic Stem Cell Expansion Upon UM171 Treatment. Blood |
| Gress V, Roussy M, Boulianne L, et al. (2023) CBFA2T3::GLIS2 Pediatric Acute Megakaryoblastic Leukemia is Sensitive to BCL-XL Inhibition by Navitoclax and DT2216. Blood Advances |
| Zhang YW, Velasco-Hernandez T, Mess J, et al. (2023) GPRC5C Drives Branched-Chain Amino Acid Metabolism in Leukemogenesis. Blood Advances |
| Moison C, Spinella JF, Chagraoui J, et al. (2022) HMGA2 expression defines a subset of AML with immature transcriptional signature and vulnerability to G2/M inhibition. Blood Advances |
| Zhang YW, Mess J, Aizarani N, et al. (2022) Hyaluronic acid-GPRC5C signalling promotes dormancy in haematopoietic stem cells. Nature Cell Biology |
| Dumont-Lagacé M, Feghaly A, Meunier MC, et al. (2022) UM171 Expansion of Cord Blood Improves Donor Availability and HLA Matching For All Patients, Including Minorities. Transplantation and Cellular Therapy |
| Ansari U, Tomellini E, Chagraoui J, et al. (2022) CEACAM1 is a novel culture-compatible surface marker of expanded long-term reconstituting hematopoietic stem cells. Blood Advances |
| He L, Arnold C, Thoma J, et al. (2022) CDK7/12/13 inhibition targets an oscillating leukemia stem cell network and synergizes with venetoclax in acute myeloid leukemia. Embo Molecular Medicine. e14990 |
| Lehnertz B, Chagraoui J, MacRae T, et al. (2021) HLF Expression Defines the Human Hematopoietic Stem Cell State. Blood |
| Herbrich S, Baran N, Cai T, et al. (2021) Overexpression of CD200 is a Stem Cell-Specific Mechanism of Immune Evasion in AML. Journal For Immunotherapy of Cancer. 9 |