Nicole D. Facompre, Ph.D.
Affiliations: | 2010 | Biochemistry and Molecular Biology | Pennsylvania State University, State College, PA, United States |
Area:
Biochemistry, Molecular Biology, OncologyGoogle:
"Nicole Facompre"Parents
Sign in to add mentor| Karam E. Bayoumy | grad student | 2010 | Penn State | |
| (Organoselenium-mediated alteration of prostate cancer cell signaling pathways.) | ||||
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Publications
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| Facompre ND, Rajagopalan P, Sahu V, et al. (2020) Identifying predictors of HPV-related head and neck squamous cell carcinoma progression and survival through patient-derived models. International Journal of Cancer |
| Facompre ND, Rajagopalan P, Pearson AT, et al. (2020) Abstract A01: HPV+ oral cancer PDXs identify prognostic utility for E2F target gene expression and mutation burden Clinical Cancer Research. 26 |
| Facompre ND, Rajagopalan P, Sahu V, et al. (2020) Abstract A03: Predictors of HPV-related head and neck cancer progression identified through patient-derived models Cancer Research. 80 |
| Basu D, Facompre ND, Gimotty PA, et al. (2019) Identifying adverse molecular features of HPV+ head and neck cancers using patient-derived models. Journal of Clinical Oncology. 37: 6057-6057 |
| Facompre ND, Harmeyer KM, Sahu V, et al. (2018) Targeting JARID1B's demethylase activity blocks a subset of its functions in oral cancer. Oncotarget. 9: 8985-8998 |
| Harmeyer KM, Facompre ND, Herlyn M, et al. (2017) JARID1 Histone Demethylases: Emerging Targets in Cancer. Trends in Cancer. 3: 713-725 |
| Facompre ND, Sahu V, Montone KT, et al. (2017) Barriers to generating PDX models of HPV-related head and neck cancer. The Laryngoscope |
| Facompre ND, Harmeyer KM, Sole X, et al. (2016) JARID1B enables transit between distinct states of the stem-like cell population in oral cancers. Cancer Research |
| Facompre N, Belden Z, Gimotty P, et al. (2015) Abstract 1517: G0-like cells support an oral cancer stem cell pool by transitioning to a JARID1B-high state Cancer Research. 75: 1517-1517 |
| Basu D, Bewley AF, Sperry SM, et al. (2013) EGFR inhibition promotes an aggressive invasion pattern mediated by mesenchymal-like tumor cells within squamous cell carcinomas. Molecular Cancer Therapeutics. 12: 2176-86 |