Janet E. Mertz
Affiliations: | University of Wisconsin, Madison, Madison, WI |
Area:
Transcriptional regulation in DNA tumor viruses and breast cancerWebsite:
https://cancerbiology.wisc.edu/bio/mertz_j.htmlGoogle:
"Janet E. Mertz"Bio:
https://mcardle.wisc.edu/who-we-are/faculty/janet-e-mertz-phd
Cross-listing: Chemistry Tree
Parents
Sign in to add mentor| Paul Berg | grad student | 1975 | Stanford (Chemistry Tree) | |
| (Deletion mutants of simian virus 40) | ||||
| John B. Gurdon | post-doc | 1975-1976 | MRC-LMB | |
Children
Sign in to add trainee| Gayle H. O'Reilly | grad student | 2000 | UW Madison (Chemistry Tree) |
| Shouhong Guang | grad student | 2004 | UW Madison (Chemistry Tree) |
| Elizabeth H. Vu | grad student | 2007 | UW Madison (Chemistry Tree) |
| Shreyasi Das | grad student | 2010 | UW Madison (Chemistry Tree) |
| Amy L. Ellis | grad student | 2010 | UW Madison (Chemistry Tree) |
| Jennifer A. Lamberski | grad student | 2010 | UW Madison (Chemistry Tree) |
| Amanda M. Esch | grad student | 2011 | UW Madison (Chemistry Tree) |
| Tawin Iempridee | grad student | 2014 | UW Madison (Chemistry Tree) |
BETA: Related publications
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Publications
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| Kraus RJ, Cordes BA, Sathiamoorthi S, et al. (2020) Reactivation of Epstein-Barr Virus By HIF-1α Requires p53. Journal of Virology |
| Kraus RJ, Yu X, Cordes BA, et al. (2017) Hypoxia-inducible factor-1α plays roles in Epstein-Barr virus's natural life cycle and tumorigenesis by inducing lytic infection through direct binding to the immediate-early BZLF1 gene promoter. Plos Pathogens. 13: e1006404 |
| Zou J, Jones RJ, Mertz JE, et al. (2017) Protein Homeostatic Modulators, a Novel Class of Cereblon-Targeting Small Molecules As Potential Clinical Candidates in Multiple Myeloma Blood. 130: 4418-4418 |
| Jones RJ, Iempridee T, Wang X, et al. (2016) Lenalidomide, Thalidomide, and Pomalidomide Reactivate the Epstein-Barr Virus Lytic Cycle Through Phosphoinositide 3-kinase Signaling and Ikaros Expression. Clinical Cancer Research : An Official Journal of the American Association For Cancer Research |
| Nawandar DM, Wang A, Makielski K, et al. (2015) Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells. Plos Pathogens. 11: e1005195 |
| Reusch JA, Nawandar DM, Wright KL, et al. (2015) Cellular differentiation regulator BLIMP1 induces Epstein-Barr virus lytic reactivation in epithelial and B cells by activating transcription from both the R and Z promoters. Journal of Virology. 89: 1731-43 |
| Iempridee T, Reusch JA, Riching A, et al. (2014) Epstein-Barr virus utilizes Ikaros in regulating its latent-lytic switch in B cells. Journal of Virology. 88: 4811-27 |
| Kenney SC, Mertz JE. (2014) Regulation of the latent-lytic switch in Epstein-Barr virus. Seminars in Cancer Biology. 26: 60-8 |
| Das S, Becker BN, Hoffmann FM, et al. (2012) Reversal of transforming growth factor-β induced epithelial-to-mesenchymal transition and the ZEB proteins. Fibrogenesis & Tissue Repair. 5: S28 |
| Ma SD, Yu X, Mertz JE, et al. (2012) An Epstein-Barr Virus (EBV) mutant with enhanced BZLF1 expression causes lymphomas with abortive lytic EBV infection in a humanized mouse model. Journal of Virology. 86: 7976-87 |