H Peter Zassenhaus
Affiliations: | Saint Louis University, St. Louis, MO, United States |
Area:
Animal Physiology Biology, Cell Biology, Molecular BiologyGoogle:
"H Zassenhaus"
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Publications
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| Bensch KG, Mott JL, Chang SW, et al. (2009) Selective mtDNA mutation accumulation results in beta-cell apoptosis and diabetes development. American Journal of Physiology. Endocrinology and Metabolism. 296: E672-80 |
| Dubec SJ, Aurora R, Zassenhaus HP. (2008) Mitochondrial DNA mutations may contribute to aging via cell death caused by peptides that induce cytochrome c release. Rejuvenation Research. 11: 611-9 |
| Bensch KG, Degraaf W, Hansen PA, et al. (2007) A transgenic model to study the pathogenesis of somatic mtDNA mutation accumulation in beta-cells. Diabetes, Obesity & Metabolism. 9: 74-80 |
| Mott JL, Zhang D, Chang SW, et al. (2006) Mitochondrial DNA mutations cause resistance to opening of the permeability transition pore. Biochimica Et Biophysica Acta. 1757: 596-603 |
| Yoshida M, Michel H, Sazanov L, et al. (2006) MITCHELL MEDAL LECTURE Biochimica Et Biophysica Acta (Bba) - Bioenergetics. 1757: 1-551 |
| Mott JL, Zhang D, Zassenhaus HP. (2005) Mitochondrial DNA mutations, apoptosis, and the misfolded protein response. Rejuvenation Research. 8: 216-26 |
| Zhang D, Mott JL, Chang SW, et al. (2005) Mitochondrial DNA mutations activate programmed cell survival in the mouse heart. American Journal of Physiology. Heart and Circulatory Physiology. 288: H2476-83 |
| Zhang D, Ezekiel UR, Chang SW, et al. (2005) Gene expression profile in dilated cardiomyopathy caused by elevated frequencies of mitochondrial DNA mutations in the mouse heart. Cardiovascular Pathology : the Official Journal of the Society For Cardiovascular Pathology. 14: 61-9 |
| Mott JL, Zhang D, Freeman JC, et al. (2004) Cardiac disease due to random mitochondrial DNA mutations is prevented by cyclosporin A. Biochemical and Biophysical Research Communications. 319: 1210-5 |
| Zhang D, Mott JL, Farrar P, et al. (2003) Mitochondrial DNA mutations activate the mitochondrial apoptotic pathway and cause dilated cardiomyopathy. Cardiovascular Research. 57: 147-57 |