Swati P. Deb
Affiliations: | Wake Forest University School of Medicine, Winston-Salem, NC, United States |
Area:
Molecular Biology, OncologyGoogle:
"Swati Deb"
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Publications
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| Vaughan CA, Singh S, Subler MA, et al. (2021) The oncogenicity of tumor-derived mutant p53 is enhanced by the recruitment of PLK3. Nature Communications. 12: 704 |
| Singh S, Vaughan CA, Rabender C, et al. (2019) DNA replication in progenitor cells and epithelial regeneration after lung injury requires the oncoprotein MDM2. Jci Insight |
| Vaughan CA, Singh S, Grossman SR, et al. (2017) Gain-of-function p53 activates multiple signaling pathways to induce oncogenicity in lung cancer cells. Molecular Oncology |
| Singh S, Vaughan CA, Frum RA, et al. (2017) Mutant p53 establishes targetable tumor dependency by promoting unscheduled replication. The Journal of Clinical Investigation |
| Vaughan CA, Pearsall I, Singh S, et al. (2016) Addiction of lung cancer cells to GOF p53 is promoted by up-regulation of epidermal growth factor receptor through multiple contacts with p53 transactivation domain and promoter. Oncotarget |
| Gadepalli VS, Deb SP, Deb S, et al. (2014) Lung cancer stem cells, p53 mutations and MDM2. Sub-Cellular Biochemistry. 85: 359-70 |
| Vaughan C, Pearsall I, Yeudall A, et al. (2014) p53: its mutations and their impact on transcription. Sub-Cellular Biochemistry. 85: 71-90 |
| Vaughan CA, Deb SP, Deb S, et al. (2014) Preferred binding of gain-of-function mutant p53 to bidirectional promoters with coordinated binding of ETS1 and GABPA to multiple binding sites. Oncotarget. 5: 417-27 |
| Frum RA, Singh S, Vaughan C, et al. (2014) The human oncoprotein MDM2 induces replication stress eliciting early intra-S-phase checkpoint response and inhibition of DNA replication origin firing. Nucleic Acids Research. 42: 926-40 |
| Singh S, Ramamoorthy M, Vaughan C, et al. (2013) Human oncoprotein MDM2 activates the Akt signaling pathway through an interaction with the repressor element-1 silencing transcription factor conferring a survival advantage to cancer cells. Cell Death and Differentiation. 20: 558-66 |