Peter Besmer
Affiliations: | Weill Cornell Medical College, New York, NY, United States |
Area:
Molecular Biology, OncologyGoogle:
"Peter Besmer"
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Publications
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| Zhang JQ, Bosbach B, Loo JK, et al. (2020) The V654A second-site KIT mutation increases tumor oncogenesis and STAT activation in a mouse model of gastrointestinal stromal tumor. Oncogene |
| Bosbach B, Rossi F, Yozgat Y, et al. (2017) Direct engagement of the PI3K pathway by mutant KIT dominates oncogenic signaling in gastrointestinal stromal tumor. Proceedings of the National Academy of Sciences of the United States of America |
| Buono M, Facchini R, Matsuoka S, et al. (2016) A dynamic niche provides Kit ligand in a stage-specific manner to the earliest thymocyte progenitors. Nature Cell Biology. 18: 157-67 |
| Cohen NA, Zeng S, Seifert AM, et al. (2015) Pharmacological Inhibition of KIT Activates MET Signaling in Gastrointestinal Stromal Tumors. Cancer Research. 75: 2061-70 |
| Ran L, Sirota I, Cao Z, et al. (2015) Combined inhibition of MAP kinase and KIT signaling synergistically destabilizes ETV1 and suppresses GIST tumor growth. Cancer Discovery. 5: 304-15 |
| Kim TS, Cavnar MJ, Cohen NA, et al. (2014) Increased KIT inhibition enhances therapeutic efficacy in gastrointestinal stromal tumor. Clinical Cancer Research : An Official Journal of the American Association For Cancer Research. 20: 2350-62 |
| Deshpande S, Bosbach B, Yozgat Y, et al. (2013) KIT receptor gain-of-function in hematopoiesis enhances stem cell self-renewal and promotes progenitor cell expansion. Stem Cells (Dayton, Ohio). 31: 1683-95 |
| Italiano A, Chen CL, Sung YS, et al. (2012) SDHA loss of function mutations in a subset of young adult wild-type gastrointestinal stromal tumors. Bmc Cancer. 12: 408 |
| Bosbach B, Deshpande S, Rossi F, et al. (2012) Imatinib resistance and microcytic erythrocytosis in a KitV558Δ;T669I/+ gatekeeper-mutant mouse model of gastrointestinal stromal tumor. Proceedings of the National Academy of Sciences of the United States of America. 109: E2276-83 |
| Chen J, Guo T, Zhang L, et al. (2012) CD133 and CD44 are universally overexpressed in GIST and do not represent cancer stem cell markers. Genes, Chromosomes & Cancer. 51: 186-95 |