Hyeong-Reh C. Kim

Affiliations: 
Wayne State University, Detroit, MI, United States 
Area:
Molecular Biology
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"Hyeong-Reh Kim"
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Publications

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Warner RB, Najy AJ, Jung YS, et al. (2020) Establishment of Structure-Function Relationship of Tissue Inhibitor of Metalloproteinase-1 for Its Interaction with CD63: Implication for Cancer Therapy. Scientific Reports. 10: 2099
Bottrell A, Meng YH, Najy AJ, et al. (2019) An oncogenic activity of PDGF-C and its splice variant in human breast cancer. Growth Factors (Chur, Switzerland). 1-15
Kessel D, Cho WJ, Rakowski J, et al. (2019) Effects of HVP Status on Responsiveness to Ionizing Radiation vs. Photodynamic Therapy in Head and Neck Cancer Cell lines. Photochemistry and Photobiology
Wasinski BD, Bonfil RD, Sohail A, et al. (2018) Abstract 2135: Complex roles of discoidin domain receptors (DDRs) in tumor growth and experimental metastasis: role of collagen I in DDR-mediated tumor growth Cancer Research. 78: 2135-2135
Bonfil RD, Sohail A, Vranić S, et al. (2018) Abstract 1070: Discoidin domain receptor 1 (DDR1): A potential suppressor of prostate cancer progression Cancer Research. 78: 1070-1070
Jung YS, Najy AJ, Huang W, et al. (2017) HPV-associated differential regulation of tumor metabolism in oropharyngeal head and neck cancer. Oncotarget. 8: 51530-51541
Najy AJ, Jung YS, Kim HC. (2016) Abstract A33: The regulation of extracellular acidosis and cell survival by the TIMP-1/CD63 axis in breast carcinoma Cancer Research. 76
Najy AJ, Dyson G, Jena BP, et al. (2015) Matriptase Activation and Shedding through PDGF-D-mediated Extracellular Acidosis. American Journal of Physiology. Cell Physiology. ajpcell.00043.2015
Hurst N, Jung Y, Snyder M, et al. (2015) Nutrient Deprivation Affects Radiation Induced Cell Killing in Human HPV-negative Cell Lines: A Potential Metabolic Target for Radioresistant Head and Neck Cancer International Journal of Radiation Oncology*Biology*Physics. 93: E540
Yong H, Kim E, Koh M, et al. (2014) Abstract 5263: Identification of a lipid raft protein that is required for H-Ras activation and breast cancer aggressiveness Cancer Research. 74: 5263-5263
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