Thomas J. McLoughlin, Ph.D.

Affiliations: 
2001 The University of Toledo 
Area:
Cell Biology, Immunology
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"Thomas McLoughlin"

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Francis X. Pizza grad student 2001 The University of Toledo
 (Neutrophil -mediated skeletal myotube injury: The contribution of reactive oxygen and nitrogen intermediates.)
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Publications

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Marino JS, Hinds TD, Potter RA, et al. (2013) Suppression of protein kinase C theta contributes to enhanced myogenesis in vitro via IRS1 and ERK1/2 phosphorylation. Bmc Cell Biology. 14: 39
McLoughlin TJ. (2010) Tax sugar, save muscle? Journal of Applied Physiology (Bethesda, Md. : 1985). 108: 3-4
McLoughlin TJ, Smith SM, DeLong AD, et al. (2009) FoxO1 induces apoptosis in skeletal myotubes in a DNA-binding-dependent manner. American Journal of Physiology. Cell Physiology. 297: C548-55
Novak ML, Billich W, Smith SM, et al. (2009) COX-2 inhibitor reduces skeletal muscle hypertrophy in mice. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 296: R1132-9
Marino JS, Tausch BJ, Dearth CL, et al. (2008) Beta2-integrins contribute to skeletal muscle hypertrophy in mice. American Journal of Physiology. Cell Physiology. 295: C1026-36
Moylan JS, Smith JD, Chambers MA, et al. (2008) TNF induction of atrogin-1/MAFbx mRNA depends on Foxo4 expression but not AKT-Foxo1/3 signaling. American Journal of Physiology. Cell Physiology. 295: C986-93
Southgate RJ, Neill B, Prelovsek O, et al. (2007) FOXO1 regulates the expression of 4E-BP1 and inhibits mTOR signaling in mammalian skeletal muscle. The Journal of Biological Chemistry. 282: 21176-86
Kuntz AR, Griffiths CM, Rankin JM, et al. (2006) Cortisol concentrations in human skeletal muscle tissue after phonophoresis with 10% hydrocortisone gel. Journal of Athletic Training. 41: 321-4
Nader GA, McLoughlin TJ, Esser KA. (2005) mTOR function in skeletal muscle hypertrophy: increased ribosomal RNA via cell cycle regulators. American Journal of Physiology. Cell Physiology. 289: C1457-65
Bastie CC, Nahlé Z, McLoughlin T, et al. (2005) FoxO1 stimulates fatty acid uptake and oxidation in muscle cells through CD36-dependent and -independent mechanisms. The Journal of Biological Chemistry. 280: 14222-9
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