Diego Altomare, Ph.D.

Affiliations: 
2010 Biomedical Science University of South Carolina, Columbia, SC 
Area:
Molecular Biology
Google:
"Diego Altomare"

Parents

Sign in to add mentor
Kim E. Creek grad student 2010 University of South Carolina
 (Smad proteins and the TGF-beta signaling pathway during in vitro progression of HPV16-immortalized human keratinocytes and Microarray gene expression profiling of exfoliated cervical cells.)
BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Abboodi F, Buckhaults P, Altomare D, et al. (2020) HPV-inactive cell populations arise from HPV16-transformed human keratinocytes after p53 knockout. Virology. 554: 9-16
Woappi Y, Altomare D, Creek KE, et al. (2020) Self-assembling 3D spheroid cultures of human neonatal keratinocytes have enhanced regenerative properties. Stem Cell Research. 49: 102048
Chavez B, Farmaki E, Zhang Y, et al. (2020) A strategy for the Identification of Paracrine Regulators of Cancer Cell Migration. Clinical and Experimental Pharmacology & Physiology
Li Y, Hodge J, Liu Q, et al. (2020) TFEB is a master regulator of tumor-associated macrophages in breast cancer. Journal For Immunotherapy of Cancer. 8
Hosseinipour M, Wan F, Altomare D, et al. (2019) HPV16-transformed human keratinocytes depend on SIX1 expression for proliferation and HPV E6/E7 gene expression. Virology. 537: 20-30
Zhang Y, Lucius MD, Altomare D, et al. (2019) Coordination Analysis of Gene Expression Points to the Relative Impact of Different Regulators During Endoplasmic Reticulum Stress. Dna and Cell Biology
Liu C, Banister CE, Weige CC, et al. (2018) PRDM1 silences stem cell-related genes and inhibits proliferation of human colon tumor organoids. Proceedings of the National Academy of Sciences of the United States of America
Batova A, Altomare D, Creek KE, et al. (2017) Englerin A induces an acute inflammatory response and reveals lipid metabolism and ER stress as targetable vulnerabilities in renal cell carcinoma. Plos One. 12: e0172632
Batova A, Altomare D, Creek KE, et al. (2017) Abstract 3219: Targeting lipid metabolism and ER stress in renal cell carcinoma by englerin A Cancer Research. 77: 3219-3219
Zhang Y, Davis C, Shah S, et al. (2016) IL-33 promotes growth and liver metastasis of colorectal cancer in mice by remodeling the tumor microenvironment and inducing angiogenesis. Molecular Carcinogenesis
See more...