Ben G. Wen, Ph.D.

Affiliations: 
2002 University of California, San Diego, La Jolla, CA 
Area:
Immunology, Cell Biology, Molecular Biology
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"Ben Wen"

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Stephen Hedrick grad student 2002 UCSD
 (Studies on the molecular mechanisms of thymocyte negative selection and the role of murine DRAK2 in T cell activation.)
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Publications

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Grünewald J, Hunt GS, Dong L, et al. (2009) Mechanistic studies of the immunochemical termination of self-tolerance with unnatural amino acids. Proceedings of the National Academy of Sciences of the United States of America. 106: 4337-42
McGargill MA, Choy C, Wen BG, et al. (2008) Drak2 regulates the survival of activated T cells and is required for organ-specific autoimmune disease. Journal of Immunology (Baltimore, Md. : 1950). 181: 7593-605
Grünewald J, Tsao ML, Perera R, et al. (2008) Immunochemical termination of self-tolerance. Proceedings of the National Academy of Sciences of the United States of America. 105: 11276-80
Friedrich ML, Wen BG, Bain G, et al. (2005) DRAK2, a lymphoid-enriched DAP kinase, regulates the TCR activation threshold during thymocyte selection. International Immunology. 17: 1379-90
McGargill MA, Wen BG, Walsh CM, et al. (2004) A deficiency in Drak2 results in a T cell hypersensitivity and an unexpected resistance to autoimmunity. Immunity. 21: 781-91
Wen BG, Pletcher MT, Warashina M, et al. (2004) Inositol (1,4,5) trisphosphate 3 kinase B controls positive selection of T cells and modulates Erk activity. Proceedings of the National Academy of Sciences of the United States of America. 101: 5604-9
Walsh CM, Wen BG, Chinnaiyan AM, et al. (1998) A role for FADD in T cell activation and development Immunity. 8: 439-449
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