Johannes S. Schlondorff, Ph.D.
Affiliations: | 2001 | Weill Cornell Medical College, New York, NY, United States |
Area:
Cell Biology, Molecular BiologyGoogle:
"Johannes Schlondorff"Parents
Sign in to add mentor| Carl P. Blobel | grad student | 2001 | Weill Cornell Medical College | |
| (Regulation of ectodomain shedding: Insights from studying tumor necrosis factor alpha convertase (TACE) and TRANCE shedding.) | ||||
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Publications
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| Feng D, Kumar M, Muntel J, et al. (2020) Phosphorylation of ACTN4 Leads to Podocyte Vulnerability and Proteinuric Glomerulosclerosis. Journal of the American Society of Nephrology : Jasn |
| Subramanian B, Chun J, Perez-Gill C, et al. (2020) FSGS-Causing INF2 Mutation Impairs Cleaved INF2 N-Fragment Functions in Podocytes. Journal of the American Society of Nephrology : Jasn |
| Stotter BR, Talbot BE, Capen DE, et al. (2020) Cosmc-dependent mucin-type O-linked glycosylation is essential for podocyte function. American Journal of Physiology. Renal Physiology |
| Dvela-Levitt M, Kost-Alimova M, Emani M, et al. (2019) Small Molecule Targets TMED9 and Promotes Lysosomal Degradation to Reverse Proteinopathy. Cell. 178: 521-535.e23 |
| Talbot BE, Vandorpe DH, Stotter BR, et al. (2019) Transmembrane insertases and N-glycosylation critically determine synthesis, trafficking and activity of the nonselective cation channel TRPC6. The Journal of Biological Chemistry |
| Subramanian B, Sun H, Yan P, et al. (2016) Mice with mutant Inf2 show impaired podocyte and slit diaphragm integrity in response to protamine-induced kidney injury. Kidney International |
| Olabisi OA, Zhang JY, VerPlank L, et al. (2015) APOL1 kidney disease risk variants cause cytotoxicity by depleting cellular potassium and inducing stress-activated protein kinases. Proceedings of the National Academy of Sciences of the United States of America |
| Barua M, Shieh E, Schlondorff J, et al. (2014) Exome sequencing and in vitro studies identified podocalyxin as a candidate gene for focal and segmental glomerulosclerosis. Kidney International. 85: 124-33 |
| Sun H, Schlondorff J, Higgs HN, et al. (2013) Inverted formin 2 regulates actin dynamics by antagonizing Rho/diaphanous-related formin signaling. Journal of the American Society of Nephrology : Jasn. 24: 917-29 |
| Sun H, Schlondorff JS, Brown EJ, et al. (2011) Rho activation of mDia formins is modulated by an interaction with inverted formin 2 (INF2). Proceedings of the National Academy of Sciences of the United States of America. 108: 2933-8 |