William D. Hastings, Ph.D.

Affiliations: 
2005 Boston University, Boston, MA, United States 
Area:
Immunology
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"William Hastings"

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Thomas L. Rothstein grad student 2005 Boston University
 (Peritoneal B-2 cells comprise a distinct population that differentiates to a B-1b phenotype.)
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Publications

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Sehgal K, Portell AJ, Ivanova EV, et al. (2020) Dynamic single-cell RNA sequencing identifies immunotherapy persister cells following PD-1 blockade. The Journal of Clinical Investigation
Hastings WD, Anderson DE, Kassam N, et al. (2009) TIM-3 is expressed on activated human CD4+ T cells and regulates Th1 and Th17 cytokines. European Journal of Immunology. 39: 2492-501
Hastings WD, Tumang JR, Behrens TW, et al. (2006) Peritoneal B-2 cells comprise a distinct B-2 cell population with B-1b-like characteristics. European Journal of Immunology. 36: 1114-23
Hastings WD, Gurdak SM, Tumang JR, et al. (2006) CD5+/Mac-1- peritoneal B cells: a novel B cell subset that exhibits characteristics of B-1 cells. Immunology Letters. 105: 90-6
Foote LC, Evans JW, Cifuni JM, et al. (2004) Interleukin-4 produces a breakdown of tolerance in vivo with autoantibody formation and tissue damage. Autoimmunity. 37: 569-77
Tumang JR, Hastings WD, Bai C, et al. (2004) Peritoneal and splenic B-1 cells are separable by phenotypic, functional, and transcriptomic characteristics. European Journal of Immunology. 34: 2158-67
Tumang JR, Negm RS, Solt LA, et al. (2002) BCR engagement induces Fas resistance in primary B cells in the absence of functional Bruton's tyrosine kinase Journal of Immunology. 168: 2712-2719
Fischer GM, Solt LA, Hastings WD, et al. (2001) Splenic and peritoneal B-1 cells differ in terms of transcriptional and proliferative features that separate peritoneal B-1 from splenic B-2 cells. Cellular Immunology. 213: 62-71
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