Pere Puigserver
Affiliations: | Dana-Farber Cancer Institute/Harvard Medical School |
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"Pere Puigserver"Children
Sign in to add trainee| Joseph T Rodgers | grad student | Dana-Farber Cancer Institute/Harvard Medical School (Chemistry Tree) |
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Publications
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| Mutlu B, Sharabi K, Sohn JH, et al. (2025) Small molecules targeting selective PCK1 and PGC-1α lysine acetylation cause anti-diabetic action through increased lactate oxidation. Cell Chemical Biology |
| Latorre-Muro P, Vitale T, Ravichandran M, et al. (2025) Chaperone-mediated insertion of mitochondrial import receptor TOM70 protects against diet-induced obesity. Nature Cell Biology |
| Mutlu B, Sharabi K, Sohn JH, et al. (2024) Small molecules targeting selective PCK1 and PGC-1α lysine acetylation cause anti-diabetic action through increased lactate oxidation. Cell Chemical Biology |
| Sohn JH, Mutlu B, Latorre-Muro P, et al. (2023) Liver mitochondrial cristae organizing protein MIC19 promotes energy expenditure and pedestrian locomotion by altering nucleotide metabolism. Cell Metabolism |
| Yook JS, Taxin ZH, Yuan B, et al. (2023) The SLC25A47 locus controls gluconeogenesis and energy expenditure. Proceedings of the National Academy of Sciences of the United States of America. 120: e2216810120 |
| Bennett CF, O'Malley KE, Perry EA, et al. (2021) Peroxisomal-derived ether phospholipids link nucleotides to respirasome assembly. Nature Chemical Biology |
| Tavares CDJ, Aigner S, Sharabi K, et al. (2020) Transcriptome-wide analysis of PGC-1α-binding RNAs identifies genes linked to glucagon metabolic action. Proceedings of the National Academy of Sciences of the United States of America |
| Mutlu B, Puigserver P. (2020) GCN5 ACETYLTRANSFERASE IN CELLULAR ENERGETIC AND METABOLIC PROCESSES. Biochimica Et Biophysica Acta. Gene Regulatory Mechanisms. 194626 |
| Luo C, Liang J, Sharabi K, et al. (2020) Obesity/type 2 diabetes-associated liver tumors are sensitive to Cyclin D1 deficiency. Cancer Research |
| Balsa E, Perry EA, Bennett CF, et al. (2020) Defective NADPH production in mitochondrial disease complex I causes inflammation and cell death. Nature Communications. 11: 2714 |