Michelle Letarte

Affiliations: 
University of Toronto, Toronto, ON, Canada 
Area:
Cell Biology, Molecular Biology
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"Michelle Letarte"
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Publications

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Cheng KH, Mariampillai A, Lee KK, et al. (2014) Histogram flow mapping with optical coherence tomography for in vivo skin angiography of hereditary hemorrhagic telangiectasia. Journal of Biomedical Optics. 19: 086015
Al-Saleh S, John PR, Letarte M, et al. (2011) Symptomatic liver involvement in neonatal hereditary hemorrhagic telangiectasia. Pediatrics. 127: e1615-20
Mei-Zahav M, Letarte M, Faughnan ME, et al. (2006) Symptomatic children with hereditary hemorrhagic telangiectasia: a pediatric center experience. Archives of Pediatrics & Adolescent Medicine. 160: 596-601
Cohen JH, Faughnan ME, Letarte M, et al. (2005) Cost comparison of genetic and clinical screening in families with hereditary hemorrhagic telangiectasia. American Journal of Medical Genetics. Part A. 137: 153-60
Chan NL, Bourdeau A, Vera S, et al. (2004) Umbilical vein and placental vessels from newborns with hereditary haemorrhagic telangiectasia type 1 genotype are normal despite reduced expression of endoglin. Placenta. 25: 208-17
Cho SK, Bourdeau A, Letarte M, et al. (2001) Expression and function of CD105 during the onset of hematopoiesis from Flk1(+) precursors. Blood. 98: 3635-42
Bourdeau A, Faughnan ME, McDonald ML, et al. (2001) Potential role of modifier genes influencing transforming growth factor-beta1 levels in the development of vascular defects in endoglin heterozygous mice with hereditary hemorrhagic telangiectasia. The American Journal of Pathology. 158: 2011-20
Bourdeau A, Faughnan ME, Letarte M. (2000) Endoglin-deficient mice, a unique model to study hereditary hemorrhagic telangiectasia. Trends in Cardiovascular Medicine. 10: 279-85
Matsubara S, Bourdeau A, terBrugge KG, et al. (2000) Analysis of endoglin expression in normal brain tissue and in cerebral arteriovenous malformations. Stroke; a Journal of Cerebral Circulation. 31: 2653-60
Bourdeau A, Cymerman U, Paquet ME, et al. (2000) Endoglin expression is reduced in normal vessels but still detectable in arteriovenous malformations of patients with hereditary hemorrhagic telangiectasia type 1. The American Journal of Pathology. 156: 911-23
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