William P. Schiemann

Affiliations: 
University of Colorado Anschutz Medical Campus, Denver, Aurora, CO 
Area:
Pharmacology, Oncology, Cell Biology
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"William Schiemann"
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Publications

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Tian M, Schiemann WP. (2017) TGF-β Stimulation of EMT Programs Elicits Non-genomic ER-α Activity and Anti-estrogen Resistance in Breast Cancer Cells. Journal of Cancer Metastasis and Treatment. 3: 150-160
Morrison CD, Allington TM, Thompson CL, et al. (2016) c-Abl inhibits breast cancer tumorigenesis through reactivation of p53-mediated p21 expression. Oncotarget
Gupta S, Hau AM, Al-Ahmadie HA, et al. (2016) Transforming Growth Factor-β Is an Upstream Regulator of Mammalian Target of Rapamycin Complex 2-Dependent Bladder Cancer Cell Migration and Invasion. The American Journal of Pathology
Janyasupab M, Lee YH, Zhang Y, et al. (2015) Detection of Lysyl Oxidase-Like 2 (LOXL2), a Biomarker of Metastasis from Breast Cancers Using Human Blood Samples. Recent Patents On Biomarkers. 5: 93-100
Thomas DG, Yenepalli A, Denais CM, et al. (2015) Non-muscle myosin IIB is critical for nuclear translocation during 3D invasion. The Journal of Cell Biology
Correa D, Somoza RA, Lin P, et al. (2015) Mesenchymal Stem Cells regulate Melanoma Cancer Cells extravasation to bone and liver at their perivascular niche. International Journal of Cancer. Journal International Du Cancer
Peiris PM, Deb P, Doolittle E, et al. (2015) Vascular Targeting of a Gold Nanoparticle to Breast Cancer Metastasis. Journal of Pharmaceutical Sciences. 104: 2600-10
Parvani JG, Gujrati MD, Mack MA, et al. (2015) Silencing β3 Integrin by Targeted ECO/siRNA Nanoparticles Inhibits EMT and Metastasis of Triple-Negative Breast Cancer. Cancer Research. 75: 2316-25
Parvani JG, Davuluri G, Wendt MK, et al. (2015) Deptor enhances triple-negative breast cancer metastasis and chemoresistance through coupling to survivin expression. Neoplasia (New York, N.Y.). 17: 317-28
Wendt MK, Williams WK, Pascuzzi PE, et al. (2015) The antitumorigenic function of EGFR in metastatic breast cancer is regulated by expression of Mig6. Neoplasia (New York, N.Y.). 17: 124-33
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