Claudio J. Conti
Affiliations: | The University of Texas Graduate School of Biomedical Sciences at Houston |
Area:
Molecular Biology, OncologyGoogle:
"Claudio Conti"Children
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Publications
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| Zheng Y, Luo L, Lambertz IU, et al. (2022) Early Dietary Exposures Epigenetically Program Mammary Cancer Susceptibility through Igf1-Mediated Expansion of the Mammary Stem Cell Compartment. Cells. 11 |
| Guerrero-Aspizua S, González-Masa A, Conti CJ, et al. (2020) Humanization of Tumor Stroma by Tissue Engineering as a Tool to Improve Squamous Cell Carcinoma Xenograft. International Journal of Molecular Sciences. 21 |
| Gunaratna RT, Santos A, Luo L, et al. (2019) Dynamic role of the codon 72 p53 single-nucleotide polymorphism in mammary tumorigenesis in a humanized mouse model. Oncogene |
| Perez CJ, Mecklenburg L, Jaubert J, et al. (2015) Increased Susceptibility to Skin Carcinogenesis Associated with a Spontaneous Mouse Mutation in the Palmitoyl Transferase Zdhhc13 Gene. The Journal of Investigative Dermatology |
| Guerrero-Aspizua S, Larcher F, Del Río M, et al. (2015) Tumor initiation by skin Ha-ras-ment. Experimental Dermatology. 24: 252-3 |
| De Angel RE, Conti CJ, Wheatley KE, et al. (2013) The enhancing effects of obesity on mammary tumor growth and Akt/mTOR pathway activation persist after weight loss and are reversed by RAD001. Molecular Carcinogenesis. 52: 446-58 |
| Perez C, Parker-Thornburg J, Mikulec C, et al. (2012) SKHIN/Sprd, a new genetically defined inbred hairless mouse strain for UV-induced skin carcinogenesis studies. Experimental Dermatology. 21: 217-20 |
| Tian J, Berton TR, Shirley SH, et al. (2012) Developmental stage determines estrogen receptor alpha expression and non-genomic mechanisms that control IGF-1 signaling and mammary proliferation in mice Journal of Clinical Investigation. 122: 192-204 |
| Benavides F, Perez C, Blando J, et al. (2012) Protective role of cathepsin L in mouse skin carcinogenesis Molecular Carcinogenesis. 51: 352-361 |
| Blando JM, Carbajal S, Abel E, et al. (2011) Cooperation between Stat3 and Akt signaling leads to prostate tumor development in transgenic mice. Neoplasia (New York, N.Y.). 13: 254-65 |