Brigitte T. Huber
Affiliations: | Cellular & Molecular Physiology | Sackler School of Graduate Biomedical Sciences (Tufts University) |
Area:
Cell Biology, Molecular BiologyGoogle:
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Publications
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| Weindel CG, Richey LJ, Mehta AJ, et al. (2016) Autophagy in Dendritic Cells and B Cells Is Critical for the Inflammatory State of TLR7-Mediated Autoimmunity. Journal of Immunology (Baltimore, Md. : 1950) |
| Weindel CG, Richey LJ, Bolland S, et al. (2015) B cell autophagy mediates TLR7-dependent autoimmunity and inflammation. Autophagy. 11: 1010-24 |
| Vu DM, Tai A, Tatro JB, et al. (2014) γδT cells are prevalent in the proximal aorta and drive nascent atherosclerotic lesion progression and neutrophilia in hypercholesterolemic mice. Plos One. 9: e109416 |
| Mele DA, Sampson JF, Huber BT. (2011) Th17 differentiation is the default program for DPP2-deficient T-cell differentiation. European Journal of Immunology. 41: 1583-93 |
| Moriyama M, Fukuhara T, Britschgi M, et al. (2011) Complement receptor 2 is expressed in neural progenitor cells and regulates adult hippocampal neurogenesis. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 3981-9 |
| Kulik L, Chen K, Huber BT, et al. (2011) Human complement receptor type 2 (CR2/CD21) transgenic mice provide an in vivo model to study immunoregulatory effects of receptor antagonists. Molecular Immunology. 48: 883-94 |
| Danilov AV, Danilova OV, Brown JR, et al. (2010) Dipeptidyl peptidase 2 apoptosis assay determines the B-cell activation stage and predicts prognosis in chronic lymphocytic leukemia. Experimental Hematology. 38: 1167-77 |
| Danilova OV, Tai AK, Mele DA, et al. (2009) Neurogenin 3-specific dipeptidyl peptidase-2 deficiency causes impaired glucose tolerance, insulin resistance, and visceral obesity. Endocrinology. 150: 5240-8 |
| Mele DA, Bista P, Baez DV, et al. (2009) Dipeptidyl peptidase 2 is an essential survival factor in the regulation of cell quiescence. Cell Cycle (Georgetown, Tex.). 8: 2425-34 |
| Tai AK, Luka J, Ablashi D, et al. (2009) HHV-6A infection induces expression of HERV-K18-encoded superantigen. Journal of Clinical Virology : the Official Publication of the Pan American Society For Clinical Virology. 46: 47-8 |