Ferruccio Galbiati
Affiliations: | University of Pittsburgh, Pittsburgh, PA, United States |
Area:
Molecular Biology, Cell Biology, Physiology BiologyGoogle:
"Ferruccio Galbiati"
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Publications
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Volonte D, Galbiati F. (2020) Caveolin-1, a master regulator of cellular senescence. Cancer Metastasis Reviews |
Jeffries EP, Di Filippo M, Galbiati F. (2018) Failure to reabsorb the primary cilium induces cellular senescence. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. fj201801382R |
Volonte D, Vyas AR, Chen C, et al. (2017) Caveolin-1 promotes the tumor suppressor properties of oncogene-induced cellular senescence. The Journal of Biological Chemistry |
Volonte D, Zou H, Bartholomew JN, et al. (2015) Oxidative stress-induced inhibition of Sirt1 by caveolin-1 promotes p53-dependent premature senescence and stimulates the secretion of interleukin 6 (IL-6). The Journal of Biological Chemistry. 290: 4202-14 |
Peffer ME, Chandran UR, Luthra S, et al. (2014) Caveolin-1 regulates genomic action of the glucocorticoid receptor in neural stem cells. Molecular and Cellular Biology. 34: 2611-23 |
Volonte D, Liu Z, Musille PM, et al. (2013) Inhibition of nuclear factor-erythroid 2-related factor (Nrf2) by caveolin-1 promotes stress-induced premature senescence. Molecular Biology of the Cell. 24: 1852-62 |
Zou H, Volonte D, Galbiati F. (2012) Interaction of caveolin-1 with Ku70 inhibits Bax-mediated apoptosis. Plos One. 7: e39379 |
Zou H, Stoppani E, Volonte D, et al. (2011) Caveolin-1, cellular senescence and age-related diseases. Mechanisms of Ageing and Development. 132: 533-42 |
Samarasinghe RA, Di Maio R, Volonte D, et al. (2011) Nongenomic glucocorticoid receptor action regulates gap junction intercellular communication and neural progenitor cell proliferation. Proceedings of the National Academy of Sciences of the United States of America. 108: 16657-62 |
Volonte D, Galbiati F. (2011) Polymerase I and transcript release factor (PTRF)/cavin-1 is a novel regulator of stress-induced premature senescence. The Journal of Biological Chemistry. 286: 28657-61 |