Xiang-Jiao Yang

Affiliations: 
McGill University, Montreal, QC, Canada 
Area:
Molecular Biology
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"Xiang-Jiao Yang"
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Publications

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Yan K, Mousavi N, Yang XJ. (2023) Analysis of Lysine Acetylation and Acetylation-like Acylation In Vitro and In Vivo. Current Protocols. 3: e738
Latypova X, Vincent M, Mollé A, et al. (2021) Haploinsufficiency of the Sin3/HDAC corepressor complex member SIN3B causes a syndromic intellectual disability/autism spectrum disorder. American Journal of Human Genetics
Wakeling E, McEntagart M, Bruccoleri M, et al. (2021) Missense substitutions at a conserved 14-3-3 binding site in HDAC4 cause a novel intellectual disability syndrome. Hgg Advances. 2: 100015
Humbert J, Salian S, Makrythanasis P, et al. (2020) De Novo KAT5 Variants Cause a Syndrome with Recognizable Facial Dysmorphisms, Cerebellar Atrophy, Sleep Disturbance, and Epilepsy. American Journal of Human Genetics
Yan K, Rousseau J, Machol K, et al. (2020) Deficient histone H3 propionylation by BRPF1-KAT6 complexes in neurodevelopmental disorders and cancer. Science Advances. 6: eaax0021
Li L, Ghorbani M, Weisz-Hubshman M, et al. (2019) Lysine acetyltransferase 8 is involved in cerebral development and syndromic intellectual disability. The Journal of Clinical Investigation
Li L, Jin J, Yang XJ. (2019) Histone Deacetylase 3 Governs Perinatal Cerebral Development via Neural Stem and Progenitor Cells. Iscience. 20: 148-167
Li L, Jayabal S, Ghorbani M, et al. (2019) ATAT1 regulates forebrain development and stress-induced tubulin hyperacetylation. Cellular and Molecular Life Sciences : Cmls
Cogné B, Ehresmann S, Beauregard-Lacroix E, et al. (2019) Missense Variants in the Histone Acetyltransferase Complex Component Gene TRRAP Cause Autism and Syndromic Intellectual Disability. American Journal of Human Genetics
Han Z, Wu H, Kim S, et al. (2018) Revealing the Protein Propionylation Activity of the Histone Acetyltransferase Males absent on the first (MOF). The Journal of Biological Chemistry
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