Peter Gergely
Affiliations: | Semmelweis Egyetem, Hungary |
Area:
Immunology, Molecular Biology, Medicine and SurgeryGoogle:
"Peter Gergely"Children
Sign in to add trainee| Tamas Constantin | grad student | 2009 | Semmelweis Egyetem |
| Gabor Hutas | grad student | 2009 | Semmelweis Egyetem |
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Publications
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| Constantin T, Kálovics T, Ponyi A, et al. (2009) Prevalence of antiphospholipid and antinuclear antibodies in children with epilepsy. Medical Science Monitor : International Medical Journal of Experimental and Clinical Research. 15: CR164-9 |
| Kohut E, Hajdu M, Gergely P, et al. (2009) Expression of TGFbeta1 and its signaling components by peripheral lymphocytes in systemic lupus erythematosus. Pathology Oncology Research : Por. 15: 251-6 |
| Gergely P, Pullmann R, Stancato C, et al. (2005) Increased prevalence of transfusion-transmitted virus and cross-reactivity with immunodominant epitopes of the HRES-1/p28 endogenous retroviral autoantigen in patients with systemic lupus erythematosus. Clinical Immunology (Orlando, Fla.). 116: 124-34 |
| Sallai K, Nagy E, Derfalvy B, et al. (2005) Antinucleosome antibodies and decreased deoxyribonuclease activity in sera of patients with systemic lupus erythematosus. Clinical and Diagnostic Laboratory Immunology. 12: 56-9 |
| Perl A, Nagy G, Gergely P, et al. (2004) Apoptosis and mitochondrial dysfunction in lymphocytes of patients with systemic lupus erythematosus. Methods in Molecular Medicine. 102: 87-114 |
| Perl A, Gergely P, Nagy G, et al. (2004) Mitochondrial hyperpolarization: a checkpoint of T-cell life, death and autoimmunity. Trends in Immunology. 25: 360-7 |
| Hidvégi B, Nagy E, Szabó T, et al. (2003) Correlation between T-cell and mast cell activity in patients with chronic urticaria. International Archives of Allergy and Immunology. 132: 177-82 |
| Perl A, Gergely P, Puskas F, et al. (2002) Metabolic switches of T-cell activation and apoptosis. Antioxidants & Redox Signaling. 4: 427-43 |
| Puskas F, Gergely P, Niland B, et al. (2002) Differential regulation of hydrogen peroxide and Fas-dependent apoptosis pathways by dehydroascorbate, the oxidized form of vitamin C. Antioxidants & Redox Signaling. 4: 357-69 |
| Gergely P, Niland B, Gonchoroff N, et al. (2002) Persistent mitochondrial hyperpolarization, increased reactive oxygen intermediate production, and cytoplasmic alkalinization characterize altered IL-10 signaling in patients with systemic lupus erythematosus. Journal of Immunology (Baltimore, Md. : 1950). 169: 1092-101 |