Maria Shatz, Ph.D.
Affiliations: | 2007 | BIR Biological Regulation | Weizmann Institute of Science, Rehovot, Israel |
Area:
Molecular BiologyGoogle:
"Maria Shatz"Parents
Sign in to add mentor| Mordechai Liscovitch | grad student | 2007 | Weizmann Institute | |
| (Caveolin-1: Role in cancer cell survival, migration and invasiveness.) | ||||
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Publications
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| Shatz M, Shats I, Menendez D, et al. (2015) p53 amplifies Toll-like receptor 5 response in human primary and cancer cells through interaction with multiple signal transduction pathways. Oncotarget. 6: 16963-80 |
| Lowe JM, Menendez D, Bushel PR, et al. (2014) p53 and NF-κB coregulate proinflammatory gene responses in human macrophages. Cancer Research. 74: 2182-92 |
| Madenspacher JH, Azzam KM, Gowdy KM, et al. (2013) p53 Integrates host defense and cell fate during bacterial pneumonia. The Journal of Experimental Medicine. 210: 891-904 |
| Menendez D, Shatz M, Resnick MA. (2013) Interactions between the tumor suppressor p53 and immune responses. Current Opinion in Oncology. 25: 85-92 |
| Shatz M, Menendez D, Resnick MA. (2012) The human TLR innate immune gene family is differentially influenced by DNA stress and p53 status in cancer cells. Cancer Research. 72: 3948-57 |
| Menendez D, Shatz M, Azzam K, et al. (2011) The Toll-like receptor gene family is integrated into human DNA damage and p53 networks. Plos Genetics. 7: e1001360 |
| Shatz M, Lustig G, Reich R, et al. (2010) Caveolin-1 mutants P132L and Y14F are dominant negative regulators of invasion, migration and aggregation in H1299 lung cancer cells. Experimental Cell Research. 316: 1748-62 |
| Shatz M, Liscovitch M. (2008) Caveolin-1: a tumor-promoting role in human cancer. International Journal of Radiation Biology. 84: 177-89 |
| Burgermeister E, Xing X, Röcken C, et al. (2007) Differential expression and function of caveolin-1 in human gastric cancer progression. Cancer Research. 67: 8519-26 |
| Milyavsky M, Shats I, Cholostoy A, et al. (2007) Inactivation of myocardin and p16 during malignant transformation contributes to a differentiation defect. Cancer Cell. 11: 133-46 |