Karen R. Kritsch, Ph.D.

Affiliations: 
2000 University of Wisconsin, Madison, Madison, WI 
Area:
Nutrition, Molecular Biology
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"Karen Kritsch"

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Denise M. Ney grad student 2000 UW Madison
 (Regulation of the hepatic insulin -like growth factor -I system by nutrition and dexamethasone.)
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Publications

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Kritsch KR, Murali S, Adamo ML, et al. (2005) Hypoenergetic high-carbohydrate or high-fat parenteral nutrition induces a similar metabolic response with differential effects on hepatic IGF-I mRNA in dexamethasone-treated rats. The Journal of Nutrition. 135: 479-85
Kritsch KR, Murali S, Adamo ML, et al. (2002) Dexamethasone decreases serum and liver IGF-I and maintains liver IGF-I mRNA in parenterally fed rats. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 282: R528-36
Gillingham MB, Kritsch KR, Murali SG, et al. (2001) Resection upregulates the IGF-I system of parenterally fed rats with jejunocolic anastomosis. American Journal of Physiology. Gastrointestinal and Liver Physiology. 281: G1158-68
Gillingham MB, Dahly EM, Carey HV, et al. (2000) Differential jejunal and colonic adaptation due to resection and IGF-I in parenterally fed rats. American Journal of Physiology. Gastrointestinal and Liver Physiology. 278: G700-9
Kritsch KR, Huss DJ, Ney DM. (2000) Greater potency of IGF-I than IGF-I/BP-3 complex in catabolic parenterally fed rats. American Journal of Physiology. Endocrinology and Metabolism. 278: E252-62
Ney DM, Huss DJ, Gillingham MB, et al. (1999) Investigation of Insulin-Like Growth Factor (IGF)-I and Insulin Receptor Binding and Expression in Jejunum of Parenterally Fed Rats Treated with IGF-I or Growth Hormone1. Endocrinology. 140: 4850-4860
Ney DM, Huss DJ, Gillingham MB, et al. (1999) Investigation of insulin-like growth factor (IGF)-I and insulin receptor binding and expression in jejunum of parenterally fed rats treated with IGF-I or growth hormone. Endocrinology. 140: 4850-60
Lo HC, Hirvonen MD, Kritsch KR, et al. (1997) Growth hormone or insulin-like growth factor I increases fat oxidation and decreases protein oxidation without altering energy expenditure in parenterally fed rats. The American Journal of Clinical Nutrition. 65: 1384-90
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