Da-Qing Yang

Affiliations: 
University of South Dakota, Vermillion, SD, United States 
Area:
Oncology, Molecular Biology
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"Da-Qing Yang"
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Publications

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Cao W, Shen R, Richard S, et al. (2022) Inhibition of triple‑negative breast cancer proliferation and motility by reactivating p53 and inhibiting overactivated Akt. Oncology Reports. 47
Harris BRE, Zhang Y, Tao J, et al. (2021) ATM inhibitor KU-55933 induces apoptosis and inhibits motility by blocking GLUT1-mediated glucose uptake in aggressive cancer cells with sustained activation of Akt. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 35: e21264
Harris BRE, Wang D, Zhang Y, et al. (2018) Induction of the p53 tumor suppressor in cancer cells through inhibition of cap-dependent translation. Molecular and Cellular Biology
Ji B, Harris BR, Liu Y, et al. (2017) Targeting IRES-Mediated p53 Synthesis for Cancer Diagnosis and Therapeutics. International Journal of Molecular Sciences. 18
Halaby MJ, Harris BR, Miskimins WK, et al. (2015) Deregulation of IRES-mediated p53 translation in cancer cells with defective p53 response to DNA damage. Molecular and Cellular Biology
Halaby MJ, Li Y, Harris BR, et al. (2015) Translational Control Protein 80 Stimulates IRES-Mediated Translation of p53 mRNA in Response to DNA Damage. Biomed Research International. 2015: 708158
Halaby MJ, Kastein BK, Yang DQ. (2013) Chloroquine stimulates glucose uptake and glycogen synthase in muscle cells through activation of Akt. Biochemical and Biophysical Research Communications. 435: 708-13
Li Y, Xiong H, Yang DQ. (2012) Functional switching of ATM: sensor of DNA damage in proliferating cells and mediator of Akt survival signal in post-mitotic human neuron-like cells. Chinese Journal of Cancer. 31: 364-72
Yang DQ, Halaby MJ, Li Y, et al. (2011) Cytoplasmic ATM protein kinase: an emerging therapeutic target for diabetes, cancer and neuronal degeneration. Drug Discovery Today. 16: 332-8
Li Y, Yang DQ. (2010) The ATM inhibitor KU-55933 suppresses cell proliferation and induces apoptosis by blocking Akt in cancer cells with overactivated Akt. Molecular Cancer Therapeutics. 9: 113-25
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