Beate Schwer

Affiliations: 
Weill Cornell Medical College, New York, NY, United States 
Area:
Molecular Biology, Genetics
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"Beate Schwer"
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Publications

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Ghosh S, Dantuluri S, Jacewicz A, et al. (2024) Characterization of tRNA splicing enzymes RNA ligase and tRNA 2'-phosphotransferase from the pathogenic fungi . Rna (New York, N.Y.)
Bednor L, Sanchez AM, Garg A, et al. (2023) Genetic suppressor screen identifies Tgp1 (glycerophosphocholine transporter), Kcs1 (IP kinase), and Plc1 (phospholipase C) as determinants of inositol pyrophosphate toxicosis in fission yeast. Mbio. e0306223
Sanchez AM, Schwer B, Jork N, et al. (2023) Activities, substrate specificity, and genetic interactions of fission yeast Siw14, a cysteinyl-phosphatase-type inositol pyrophosphatase. Mbio. e0205623
Garg A, Schwer B, Shuman S. (2023) Fission yeast poly(A) polymerase active site mutation Y86D alleviates the synthetic growth defect and upregulates mRNAs targeted by MTREC and Mmi1. Rna (New York, N.Y.)
Sanchez AM, Garg A, Schwer B, et al. (2023) Duf89 abets lncRNA control of fission yeast phosphate homeostasis via its antagonism of precocious lncRNA transcription termination. Rna (New York, N.Y.)
Garg A, Sanchez AM, Miele M, et al. (2023) Cellular responses to long-term phosphate starvation of fission yeast: Maf1 determines fate choice between quiescence and death associated with aberrant tRNA biogenesis. Nucleic Acids Research
Benjamin B, Goldgur Y, Jork N, et al. (2022) Structures of Fission Yeast Inositol Pyrophosphate Kinase Asp1 in Ligand-Free, Substrate-Bound, and Product-Bound States. Mbio. e0308722
Benjamin B, Garg A, Jork N, et al. (2022) Activities and Structure-Function Analysis of Fission Yeast Inositol Pyrophosphate (IPP) Kinase-Pyrophosphatase Asp1 and Its Impact on Regulation of Gene Expression. Mbio. e0103422
Schwer B, Garg A, Sanchez AM, et al. (2022) Cleavage-Polyadenylation Factor Cft1 and SPX Domain Proteins Are Agents of Inositol Pyrophosphate Toxicosis in Fission Yeast. Mbio. e0347621
Garg A, Shuman S, Schwer B. (2021) Genetic screen for suppression of transcriptional interference reveals fission yeast 14-3-3 protein Rad24 as an antagonist of precocious Pol2 transcription termination. Nucleic Acids Research
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