Yedidya Saiman, Ph.D.

Affiliations: 
2012 Biomedical Sciences Mount Sinai School of Medicine, New York, NY 
Area:
Cell Biology, Immunology
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"Yedidya Saiman"

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Meena B. Bansal grad student 2012 Mount Sinai
 (The Role of Stellate Cell-derived CXCL12 in Liver Injury.)
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Publications

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Mosoian A, Zhang L, Hong F, et al. (2016) HIV infection of Kupffer cells results in an amplified proinflammatory response to LPS. Journal of Leukocyte Biology
Saiman Y, Sugiyama T, Simchoni N, et al. (2015) Biliary Epithelial Cells Are Not the Predominant Source of Hepatic CXCL12. The American Journal of Pathology. 185: 1859-66
Saiman Y, Jiao J, Fiel MI, et al. (2015) Inhibition of the CXCL12/CXCR4 chemokine axis with AMD3100, a CXCR4 small molecule inhibitor, worsens murine hepatic injury. Hepatology Research : the Official Journal of the Japan Society of Hepatology. 45: 794-803
Saiman Y, Agarwal R, Hickman DA, et al. (2013) CXCL12 induces hepatic stellate cell contraction through a calcium-independent pathway. American Journal of Physiology. Gastrointestinal and Liver Physiology. 305: G375-82
Fausther M, Sheung N, Saiman Y, et al. (2012) Activated hepatic stellate cells upregulate transcription of ecto-5'-nucleotidase/CD73 via specific SP1 and SMAD promoter elements. American Journal of Physiology. Gastrointestinal and Liver Physiology. 303: G904-14
Hong F, Saiman Y, Si C, et al. (2012) X4 Human immunodeficiency virus type 1 gp120 promotes human hepatic stellate cell activation and collagen I expression through interactions with CXCR4. Plos One. 7: e33659
Radbill BD, Gupta R, Ramirez MC, et al. (2011) Loss of matrix metalloproteinase-2 amplifies murine toxin-induced liver fibrosis by upregulating collagen I expression. Digestive Diseases and Sciences. 56: 406-16
Tuyama AC, Hong F, Saiman Y, et al. (2010) Human immunodeficiency virus (HIV)-1 infects human hepatic stellate cells and promotes collagen I and monocyte chemoattractant protein-1 expression: implications for the pathogenesis of HIV/hepatitis C virus-induced liver fibrosis. Hepatology (Baltimore, Md.). 52: 612-22
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