David E. Levin
Affiliations: | Biochemistry | Johns Hopkins University, Baltimore, MD |
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"David Levin"Children
Sign in to add traineeKyung S. Lee | grad student | Johns Hopkins University Schoolof Public Health (Chemistry Tree) | |
Andrew K. Sobering | grad student | 2001 | Johns Hopkins |
Bevin A. Philip | grad student | 2002 | Johns Hopkins |
Martin J. Romeo | grad student | 2004 | Johns Hopkins |
Sara E. Beese | grad student | 2011 | Johns Hopkins |
Andrew Truman | post-doc | 2006-2010 | Johns Hopkins |
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Publications
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Liu L, Veis J, Reiter W, et al. (2021) Regulation of Pkc1 Hyper-Phosphorylation by Genotoxic Stress. Journal of Fungi (Basel, Switzerland). 7 |
Laz EV, Lee J, Levin DE. (2020) Crosstalk between Saccharomycescerevisiae SAPKs Hog1 and Mpk1 is mediated by glycerol accumulation. Fungal Biology. 124: 361-367 |
Lee J, Levin DE. (2019) Methylated metabolite of arsenite blocks glycerol production in yeast by inhibition of glycerol-3-phosphate dehydrogenase. Molecular Biology of the Cell. mbcE19040228 |
Lee J, Liu L, Levin DE. (2018) Stressing out or stressing in: intracellular pathways for SAPK activation. Current Genetics |
Liu L, Levin DE. (2018) Intracellular Mechanism by which Genotoxic Stress Activates Yeast SAPK Mpk1. Molecular Biology of the Cell. mbcE18070441 |
Lee J, Levin DE. (2018) Intracellular Mechanism by which Arsenite Activates Yeast Stress MAPK Hog1. Molecular Biology of the Cell. mbcE18030185 |
Lee J, Levin DE. (2015) Rgc2 Regulator of Glycerol Channel Fps1 Functions as a Homo- and Heterodimer with Rgc1. Eukaryotic Cell. 14: 719-25 |
Lee J, Reiter W, Dohnal I, et al. (2013) MAPK Hog1 closes the S. cerevisiae glycerol channel Fps1 by phosphorylating and displacing its positive regulators. Genes & Development. 27: 2590-601 |
Beese-Sims SE, Pan SJ, Lee J, et al. (2012) Mutants in the Candida glabrata glycerol channels are sensitized to cell wall stress. Eukaryotic Cell. 11: 1512-9 |
Levin DE. (2011) Regulation of cell wall biogenesis in Saccharomyces cerevisiae: the cell wall integrity signaling pathway. Genetics. 189: 1145-75 |