Alicia Lee

2002-2006 National Cancer Institute, NIH, Bethesda, Bethesda, MD, United States 
"Alicia Lee"
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Christiansen EM, Yang SJ, Ando DM, et al. (2018) In Silico Labeling: Predicting Fluorescent Labels in Unlabeled Images. Cell
Skibinski G, Hwang V, Ando DM, et al. (2016) Nrf2 mitigates LRRK2- and α-synuclein-induced neurodegeneration by modulating proteostasis. Proceedings of the National Academy of Sciences of the United States of America
Wang KC, Nguyen P, Weiss A, et al. (2014) MicroRNA-23b regulates cyclin-dependent kinase-activating kinase complex through cyclin H repression to modulate endothelial transcription and growth under flow. Arteriosclerosis, Thrombosis, and Vascular Biology. 34: 1437-45
Viana-Pereira M, Lee A, Popov S, et al. (2011) Microsatellite instability in pediatric high grade glioma is associated with genomic profile and differential target gene inactivation. Plos One. 6: e20588
Lee AV, Moriarty JP, Borgstrom C, et al. (2010) What can we learn from patient dissatisfaction? An analysis of dissatisfying events at an academic medical center. Journal of Hospital Medicine. 5: 514-20
Bernstein KA, Granneman S, Lee AV, et al. (2006) Comprehensive mutational analysis of yeast DEXD/H box RNA helicases involved in large ribosomal subunit biogenesis. Molecular and Cellular Biology. 26: 1195-208
Sivarao DV, Newberry K, Langdon S, et al. (2005) Effect of 4-(5-chloro-2-hydroxyphenyl)-3-(2-hydroxyethyl)-6-(trifluoromethyl)-quinolin-2(1H)-one (BMS-223131), a novel opener of large conductance Ca2+-activated K+ (maxi-K) channels on normal and stress-aggravated colonic motility and visceral nociception. The Journal of Pharmacology and Experimental Therapeutics. 313: 840-7
Lee AC, Fernandez-Capetillo O, Pisupati V, et al. (2005) Specific association of mouse MDC1/NFBD1 with NBS1 at sites of DNA-damage. Cell Cycle (Georgetown, Tex.). 4: 177-82
Fernandez-Capetillo O, Lee A, Nussenzweig M, et al. (2004) H2AX: the histone guardian of the genome. Dna Repair. 3: 959-67
Celeste A, Fernandez-Capetillo O, Kruhlak MJ, et al. (2003) Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks. Nature Cell Biology. 5: 675-9
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